Pages that link to "Q46103652"
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The following pages link to Chimeric homeobox gene E2A-PBX1 induces proliferation, apoptosis, and malignant lymphomas in transgenic mice (Q46103652):
Displaying 50 items.
- Oncogenic homeodomain transcription factor E2A-Pbx1 activates a novel WNT gene in pre-B acute lymphoblastoid leukemia (Q22010579) (← links)
- Cloning and expression of a new HOXC6 transcript encoding a repressing protein (Q24314342) (← links)
- E2A basic-helix-loop-helix transcription factors are negatively regulated by serum growth factors and by the Id3 protein (Q24316427) (← links)
- Structural determinants within Pbx1 that mediate cooperative DNA binding with pentapeptide-containing Hox proteins: proposal for a model of a Pbx1-Hox-DNA complex (Q24317529) (← links)
- The AD1 and AD2 transactivation domains of E2A are essential for the antiapoptotic activity of the chimeric oncoprotein E2A-HLF (Q24522770) (← links)
- The novel homeoprotein Prep1 modulates Pbx-Hox protein cooperativity (Q24533177) (← links)
- Fusion with E2A converts the Pbx1 homeodomain protein into a constitutive transcriptional activator in human leukemias carrying the t(1;19) translocation (Q24612257) (← links)
- DNA-binding specificity and trans-activating potential of the leukemia-associated E2A-hepatic leukemia factor fusion protein (Q24612650) (← links)
- Meis1 and pKnox1 bind DNA cooperatively with Pbx1 utilizing an interaction surface disrupted in oncoprotein E2a-Pbx1 (Q24646482) (← links)
- HOXB7 constitutively activates basic fibroblast growth factor in melanomas (Q24648665) (← links)
- Critical role for a single leucine residue in leukemia induction by E2A-PBX1 (Q24670517) (← links)
- A robust in vivo model for B cell precursor acute lymphoblastic leukemia (Q28085325) (← links)
- The leukemogenic transcription factor E2a-Pbx1 induces expression of the putative N-myc and p53 target gene NDRG1 in Ba/F3 cells (Q28203372) (← links)
- Cloning and functional characterization of MEF2D/DAZAP1 and DAZAP1/MEF2D fusion proteins created by a variant t(1;19)(q23;p13.3) in acute lymphoblastic leukemia (Q28237803) (← links)
- A conserved C-terminal domain in PBX increases DNA binding by the PBX homeodomain and is not a primary site of contact for the YPWM motif of HOXA1 (Q28270511) (← links)
- Disordered T-cell development and T-cell malignancies in SCL LMO1 double-transgenic mice: parallels with E2A-deficient mice (Q28506087) (← links)
- HoxB8 requires its Pbx-interaction motif to block differentiation of primary myeloid progenitors and of most cell line models of myeloid differentiation (Q30176411) (← links)
- Murine models of acute leukemia: important tools in current pediatric leukemia research (Q33606018) (← links)
- Genetic Modeling of PIM Proteins in Cancer: Proviral Tagging and Cooperation with Oncogenes, Tumor Suppressor Genes, and Carcinogens. (Q33646972) (← links)
- Disrupted differentiation and oncogenic transformation of lymphoid progenitors in E2A-HLF transgenic mice (Q33652525) (← links)
- High incidence of proviral integrations in the Hoxa locus in a new model of E2a-PBX1-induced B-cell leukemia. (Q33763739) (← links)
- Cytogenetics and molecular genetics of childhood leukemia (Q33820790) (← links)
- Motif-guided sparse decomposition of gene expression data for regulatory module identification (Q33852615) (← links)
- EB-1, a tyrosine kinase signal transduction gene, is transcriptionally activated in the t(1;19) subset of pre-B ALL, which express oncoprotein E2a-Pbx1. (Q33874482) (← links)
- The oncoprotein E2A-Pbx1a collaborates with Hoxa9 to acutely transform primary bone marrow cells (Q33959545) (← links)
- Role for homodimerization in growth deregulation by E2a fusion proteins (Q33964971) (← links)
- Transcription factors and translocations in lymphoid and myeloid leukemia (Q34173877) (← links)
- Biology and treatment of adult acute lymphoblastic leukemia (Q34182822) (← links)
- The role of E2A-PBX1 in leukemogenesis (Q34405471) (← links)
- E2A-HLF usurps control of evolutionarily conserved survival pathways (Q34405477) (← links)
- The molecular genetics of hematologic malignancies (Q35224361) (← links)
- E2a/Pbx1 induces the rapid proliferation of stem cell factor-dependent murine pro-T cells that cause acute T-lymphoid or myeloid leukemias in mice (Q35544255) (← links)
- Standardization and quality control studies of 'real-time' quantitative reverse transcriptase polymerase chain reaction of fusion gene transcripts for residual disease detection in leukemia - a Europe Against Cancer program. (Q35561038) (← links)
- Pre-B-cell leukemia transcription factor 1 regulates expression of valosin-containing protein, a gene involved in cancer growth (Q35570348) (← links)
- Retrovirus-Mediated Expression of E2A-PBX1 Blocks Lymphoid Fate but Permits Retention of Myeloid Potential in Early Hematopoietic Progenitors (Q35670892) (← links)
- Restricted expression of E2A protein in primary human tissues correlates with proliferation and differentiation (Q35753130) (← links)
- Cdx-2 homeodomain protein expression in human and rat colorectal adenoma and carcinoma (Q35798391) (← links)
- Transgenic mice as an in vivo model of lymphomagenesis (Q35840178) (← links)
- Conditional Expression of E2A-HLF Induces B-Cell Precursor Death and Myeloproliferative-Like Disease in Knock-In Mice (Q35847141) (← links)
- Comparative genomics reveals multistep pathogenesis of E2A-PBX1 acute lymphoblastic leukemia (Q36106034) (← links)
- Regulation of cell division cycle progression by bcl-2 expression: a potential mechanism for inhibition of programmed cell death (Q36366587) (← links)
- Crosstalk between ROR1 and the Pre-B cell receptor promotes survival of t(1;19) acute lymphoblastic leukemia (Q36406203) (← links)
- CD3-epsilon overexpressed in prothymocytes acts as an oncogene (Q36437811) (← links)
- E2A-HLF-mediated cell transformation requires both the trans-activation domains of E2A and the leucine zipper dimerization domain of HLF. (Q36551510) (← links)
- Both Pbx1 and E2A-Pbx1 bind the DNA motif ATCAATCAA cooperatively with the products of multiple murine Hox genes, some of which are themselves oncogenes. (Q36552793) (← links)
- Cooperative interactions between HOX and PBX proteins mediated by a conserved peptide motif (Q36553133) (← links)
- The Hox cooperativity motif of the chimeric oncoprotein E2a-Pbx1 is necessary and sufficient for oncogenesis (Q36564617) (← links)
- High incidence of T-cell tumors in E2A-null mice and E2A/Id1 double-knockout mice (Q36574096) (← links)
- Hox gene products modulate the DNA binding activity of Pbx1 and Pbx2 (Q36683631) (← links)
- Detection of E2A-PBX1 fusion transcripts in human non-small-cell lung cancer (Q36866974) (← links)