Pages that link to "Q43526519"
Jump to navigation
Jump to search
The following pages link to Adenovirus-mediated gene transfer of the human TIMP-1 gene inhibits smooth muscle cell migration and neointimal formation in human saphenous vein (Q43526519):
Displaying 50 items.
- Gene Therapy for Cardiovascular Disease (Q24791063) (← links)
- Gene therapy strategies to facilitate organ transplantation (Q33720575) (← links)
- Proteolysis and cell migration: creating a path? (Q33744936) (← links)
- Who are the proteolytic culprits in vascular disease? (Q33764359) (← links)
- Cardiovascular gene therapy (Q33838040) (← links)
- Current, new and future treatments in dyslipidaemia and atherosclerosis (Q33993678) (← links)
- Endothelial cell delivery for cardiovascular therapy (Q34003157) (← links)
- Therapeutic potential of matrix metalloproteinase inhibitors in atherosclerosis (Q34074837) (← links)
- Gene therapy for vein graft disease (Q34119443) (← links)
- Local gene delivery to the vessel wall (Q34418987) (← links)
- Matrix metalloproteinases in tumor invasion: role for cell migration (Q34657461) (← links)
- Understanding and treating vein graft atherosclerosis (Q34934966) (← links)
- The role of matrix metalloproteinases in vascular disease (Q34981650) (← links)
- Gene transfer to the vasculature: historical perspective and implication for future research objectives (Q34982950) (← links)
- Matrix metalloproteinases and coronary artery diseases (Q35081023) (← links)
- In Situ Zymography: A Molecular Pathology Technique to Localize Endogenous Protease Activity in Tissue Sections (Q35118613) (← links)
- The dynamic extracellular matrix: intervention strategies during heart failure and atherosclerosis (Q35170453) (← links)
- Preventing saphenous vein graft failure: does gene therapy have a role? (Q35214863) (← links)
- Temporal regulation of venous extracellular matrix components during arteriovenous fistula maturation. (Q35508544) (← links)
- Nox1 transactivation of epidermal growth factor receptor promotes N-cadherin shedding and smooth muscle cell migration (Q35766044) (← links)
- Loss of Endothelial Barrier in Marfan Mice (mgR/mgR) Results in Severe Inflammation after Adenoviral Gene Therapy (Q35913519) (← links)
- Cardiovascular gene delivery: The good road is awaiting (Q35915765) (← links)
- Integrase-deficient lentiviral vectors mediate efficient gene transfer to human vascular smooth muscle cells with minimal genotoxic risk (Q36469473) (← links)
- Tissue kallikrein is essential for invasive capacity of circulating proangiogenic cells. (Q36682256) (← links)
- Gene therapy of fibroproliferative vasculopathies: current ideas in molecular mechanisms and biomedical technology (Q36690251) (← links)
- Matrix metalloproteinases: influence on smooth muscle cells and atherosclerotic plaque stability (Q36752100) (← links)
- Possible involvement of Cyclic-GMP-dependent protein kinase on matrix metalloproteinase-2 expression in rat aortic smooth muscle cells (Q36783935) (← links)
- Matrix metalloproteinase-1 and tissue inhibitors do not predict incident coronary artery disease in the atherosclerosis risk in communities (ARIC) study (Q37023639) (← links)
- Gene therapy to prevent occlusion of venous bypass grafts (Q37176430) (← links)
- Suppression of neointima formation by targeting β-catenin/TCF pathway (Q37487719) (← links)
- Matrix metalloproteinase inhibition therapy for vascular diseases (Q37983293) (← links)
- Vein graft failure: current clinical practice and potential for gene therapeutics (Q37997898) (← links)
- Gene therapy for cardiovascular disease: perspectives and potential (Q38059008) (← links)
- Smooth muscle cell hypertrophy, proliferation, migration and apoptosis in pulmonary hypertension (Q38112038) (← links)
- Coronary artery bypass graft: why is the saphenous vein prone to intimal hyperplasia? (Q38220601) (← links)
- Vein graft failure: from pathophysiology to clinical outcomes. (Q38837879) (← links)
- Efficient transduction of primary vascular cells by the rare adenovirus serotype 49 vector (Q38901143) (← links)
- TIMP-1 modulates chemotaxis of human neural stem cells through CD63 and integrin signalling (Q39014784) (← links)
- Critical role of tissue kallikrein in vessel formation and maturation: implications for therapeutic revascularization. (Q39248182) (← links)
- Over‐expression of mitogen‐activated protein kinase phosphatase‐2 enhances adhesion molecule expression and protects against apoptosis in human endothelial cells (Q39835063) (← links)
- Regulation of cell-matrix contacts and beta-catenin signaling in VSMC by integrin-linked kinase: implications for intimal thickening (Q39999153) (← links)
- Gene-therapy delivery strategies in cardiology (Q40119187) (← links)
- Targeting Wnt/β-Catenin Activated Cells with Dominant-Negative N-cadherin to Reduce Neointima Formation. (Q40187241) (← links)
- Adeno-associated virus (AAV)-7 and -8 poorly transduce vascular endothelial cells and are sensitive to proteasomal degradation. (Q40411990) (← links)
- Preventing intimal thickening of vein grafts in vein artery bypass using STAT-3 siRNA. (Q41531960) (← links)
- Pseudotyping the adenovirus serotype 5 capsid with both the fibre and penton of serotype 35 enhances vascular smooth muscle cell transduction. (Q41765546) (← links)
- Inhibition of N-cadherin retards smooth muscle cell migration and intimal thickening via induction of apoptosis (Q42177448) (← links)
- Inhibition of invasion and induction of apoptotic cell death of cancer cell lines by overexpression of TIMP-3. (Q42280122) (← links)
- Remodeling of the vascular tunica media is essential for development of collateral vessels in the canine heart (Q42470540) (← links)
- Potent inhibition of arterial intimal hyperplasia by TIMP1 gene transfer using AAV vectors (Q43682973) (← links)