Pages that link to "Q39724660"

The following pages link to Rem, a member of the RGK GTPases, inhibits recombinant CaV1.2 channels using multiple mechanisms that require distinct conformations of the GTPase (Q39724660):

Displaying 45 items.

• Stac3 is a component of the excitation-contraction coupling machinery and mutated in Native American myopathy (Q24294570) (← links)
• L-type calcium channel targeting and local signalling in cardiac myocytes (Q26853101) (← links)
• Structure-function of proteins interacting with the α1 pore-forming subunit of high-voltage-activated calcium channels (Q26864613) (← links)
• Small G proteins in the cardiovascular system: physiological and pathological aspects (Q27010607) (← links)
• BARP suppresses voltage-gated calcium channel activity and Ca2+-evoked exocytosis (Q28512209) (← links)
• Homodimerization of the Src homology 3 domain of the calcium channel β-subunit drives dynamin-dependent endocytosis. (Q30155579) (← links)
• The β Subunit of Voltage-Gated Ca2+Channels (Q30155988) (← links)
• Direct inhibition of P/Q-type voltage-gated Ca 2+ channels by Gem does not require a direct Gem/Ca v β interaction (Q30433191) (← links)
• A loss-of-function analysis reveals that endogenous Rem2 promotes functional glutamatergic synapse formation and restricts dendritic complexity (Q31134091) (← links)
• Ancient origins of RGK protein function: modulation of voltage-gated calcium channels preceded the protostome and deuterostome split. (Q31172439) (← links)
• Differential effects of RGK proteins on L-type channel function in adult mouse skeletal muscle (Q33595731) (← links)
• Regulation of high-voltage-activated Ca(2+) channel function, trafficking, and membrane stability by auxiliary subunits (Q33763700) (← links)
• Progress in the structural understanding of voltage-gated calcium channel (CaV) function and modulation (Q33765534) (← links)
• Gαi2- and Gαi3-specific regulation of voltage-dependent L-type calcium channels in cardiomyocytes (Q34037885) (← links)
• Molecular mechanisms, and selective pharmacological rescue, of Rem-inhibited CaV1.2 channels in heart (Q34187371) (← links)
• Distinct RGK GTPases differentially use α1- and auxiliary β-binding-dependent mechanisms to inhibit CaV1.2/CaV2.2 channels (Q34271168) (← links)
• Bio-inspired voltage-dependent calcium channel blockers (Q34308192) (← links)
• LQT1 mutations in KCNQ1 C-terminus assembly domain suppress IKs using different mechanisms (Q34974496) (← links)
• Solution NMR and calorimetric analysis of Rem2 binding to the Ca2+ channel β4 subunit: a low affinity interaction is required for inhibition of Cav2.1 Ca2+ currents (Q35555097) (← links)
• Rem uncouples excitation-contraction coupling in adult skeletal muscle fibers. (Q35795789) (← links)
• Molecular determinants of Gem protein inhibition of P/Q-type Ca2+ channels (Q36080574) (← links)
• Rem-GTPase regulates cardiac myocyte L-type calcium current (Q36201581) (← links)
• Similar molecular determinants on Rem mediate two distinct modes of inhibition of CaV1.2 channels (Q37183263) (← links)
• Manipulating L-type calcium channels in cardiomyocytes using split-intein protein transsplicing (Q37192045) (← links)
• Regulation of voltage-dependent calcium channels by RGK proteins (Q38051949) (← links)
• Ion channel engineering: perspectives and strategies (Q38247898) (← links)
• RGK regulation of voltage-gated calcium channels (Q38313471) (← links)
• Functional assessment of three Rem residues identified as critical for interactions with Ca(2+) channel β subunits. (Q38899941) (← links)
• Quantifying macromolecular interactions in living cells using FRET two-hybrid assays (Q39199394) (← links)
• Rad and Rem are non-canonical G-proteins with respect to the regulatory role of guanine nucleotide binding in Ca(V)1.2 channel regulation. (Q40478387) (← links)
• Defining the MO's of RGK proteins (Q41711413) (← links)
• Adrenergic signaling controls RGK-dependent trafficking of cardiac voltage-gated L-type Ca2+ channels through PKD1. (Q41917870) (← links)
• Mechanism of auxiliary β-subunit-mediated membrane targeting of L-type (Ca(V)1.2) channels (Q46875861) (← links)
• Sculpting ion channel functional expression with engineered ubiquitin ligases. (Q47277481) (← links)
• Molecular mechanisms and physiological relevance of RGK proteins in the heart (Q47297180) (← links)
• Impaired excitation-contraction coupling in muscle fibres from the dynamin2R465W mouse model of centronuclear myopathy. (Q47601966) (← links)
• Bilobal architecture is a requirement for calmodulin signaling to CaV1.3 channels. (Q52659621) (← links)
• Development of a High-Throughput Flow Cytometry Assay to Monitor Defective Trafficking and Rescue of Long QT2 Mutant hERG Channels. (Q55362439) (← links)
• Engineering selectivity into RGK GTPase inhibition of voltage-dependent calcium channels (Q58612164) (← links)
• Regulation of microdomain voltage-gated L-type calcium channels in cardiac health and disease. (Q64941811) (← links)
• A potent voltage-gated calcium channel inhibitor engineered from a nanobody targeted to auxiliary Caβ subunits (Q83230197) (← links)
• Rem-induced inhibition of Ca2+ channels--a three-pronged assault (Q84338611) (← links)
• Duplex signaling by CaM and Stac3 enhances CaV1.1 function and provides insights into congenital myopathy (Q89294511) (← links)
• Designer genetically-encoded voltage-dependent calcium channel inhibitors inspired by RGK GTPases (Q89913108) (← links)
• Dopamine transporter trafficking and Rit2 GTPase: Mechanism of action and in vivo impact (Q90051289) (← links)