Pages that link to "Q37028275"
Jump to navigation
Jump to search
The following pages link to Structural variation in the glycan strands of bacterial peptidoglycan (Q37028275):
Displaying 50 items.
- Masquerading microbial pathogens: capsular polysaccharides mimic host-tissue molecules (Q26865784) (← links)
- Colonize, evade, flourish: how glyco-conjugates promote virulence of Helicobacter pylori (Q26866466) (← links)
- Peptidoglycan remodeling by the coordinated action of multispecific enzymes (Q27011518) (← links)
- P. aeruginosa SGNH Hydrolase-Like Proteins AlgJ and AlgX Have Similar Topology but Separate and Distinct Roles in Alginate Acetylation (Q27640580) (← links)
- Identification, Structure, and Function of a Novel Type VI Secretion Peptidoglycan Glycoside Hydrolase Effector-Immunity Pair (Q27679179) (← links)
- Structure of MurNAc 6-Phosphate Hydrolase (MurQ) from Haemophilus influenzae with a Bound Inhibitor (Q27680647) (← links)
- The elucidation of the structure of Thermotoga maritima peptidoglycan reveals two novel types of cross-link (Q28249287) (← links)
- The peptidoglycan sacculus of Myxococcus xanthus has unusual structural features and is degraded during glycerol-induced myxospore development (Q28300374) (← links)
- Characterization of an N-acetylmuramic acid/N-acetylglucosamine kinase of Clostridium acetobutylicum (Q28485893) (← links)
- Turning defense into offense: defensin mimetics as novel antibiotics targeting lipid II (Q28534946) (← links)
- Biochemical and biophysical characterization of PlyGRCS, a bacteriophage endolysin active against methicillin-resistant Staphylococcus aureus (Q30009348) (← links)
- Synthase-dependent exopolysaccharide secretion in Gram-negative bacteria (Q30155195) (← links)
- Resistance to mucosal lysozyme compensates for the fitness deficit of peptidoglycan modifications by Streptococcus pneumoniae (Q33392682) (← links)
- Peptidoglycan O-acetylation increases in response to vancomycin treatment in vancomycin-resistant Enterococcus faecalis (Q33559903) (← links)
- How Listeria monocytogenes organizes its surface for virulence (Q33569229) (← links)
- Biosynthesis of a new UDP-sugar, UDP-2-acetamido-2-deoxyxylose, in the human pathogen Bacillus cereus subspecies cytotoxis NVH 391-98. (Q33597259) (← links)
- From the regulation of peptidoglycan synthesis to bacterial growth and morphology. (Q33694168) (← links)
- Staphylococcus haemolyticus prophage ΦSH2 endolysin relies on cysteine, histidine-dependent amidohydrolases/peptidases activity for lysis 'from without' (Q33775374) (← links)
- The bacterial cell envelope (Q33800394) (← links)
- Daughter cell separation is controlled by cytokinetic ring-activated cell wall hydrolysis (Q33845937) (← links)
- Host-guest chemistry of the peptidoglycan (Q34013593) (← links)
- Phagocytosis and phagosome acidification are required for pathogen processing and MyD88-dependent responses to Staphylococcus aureus (Q34112627) (← links)
- Cell wall structure and function in lactic acid bacteria (Q34139858) (← links)
- Both TLR2 and TRIF contribute to interferon-β production during Listeria infection (Q34202860) (← links)
- Peptidoglycan-modifying enzyme Pgp1 is required for helical cell shape and pathogenicity traits in Campylobacter jejuni (Q34211534) (← links)
- The sweet tooth of bacteria: common themes in bacterial glycoconjugates. (Q34297837) (← links)
- Bacteriophage endolysins as novel antimicrobials (Q34303439) (← links)
- Regulation of neuraminidase expression in Streptococcus pneumoniae. (Q34410365) (← links)
- Immune receptors involved in Streptococcus suis recognition by dendritic cells (Q34416075) (← links)
- Listeria monocytogenes is resistant to lysozyme through the regulation, not the acquisition, of cell wall-modifying enzymes (Q34435153) (← links)
- Dual role for the O-acetyltransferase OatA in peptidoglycan modification and control of cell septation in Lactobacillus plantarum (Q34460694) (← links)
- The peptidoglycan of Mycobacterium abscessus is predominantly cross-linked by L,D-transpeptidases (Q34491657) (← links)
- Modifications to the peptidoglycan backbone help bacteria to establish infection (Q34529521) (← links)
- O-Acetylation of peptidoglycan is required for proper cell separation and S-layer anchoring in Bacillus anthracis (Q34568418) (← links)
- A phyletically rare gene promotes the niche-specific fitness of an E. coli pathogen during bacteremia. (Q34607973) (← links)
- Growth medium-dependent glycine incorporation into the peptidoglycan of Caulobacter crescentus (Q34612262) (← links)
- Lysozymes in the animal kingdom (Q34659960) (← links)
- Characterization of O-acetylation of N-acetylglucosamine: a novel structural variation of bacterial peptidoglycan (Q35085058) (← links)
- Nod2 sensing of lysozyme-digested peptidoglycan promotes macrophage recruitment and clearance of S. pneumoniae colonization in mice. (Q35187119) (← links)
- Mutations of the Listeria monocytogenes peptidoglycan N-deacetylase and O-acetylase result in enhanced lysozyme sensitivity, bacteriolysis, and hyperinduction of innate immune pathways (Q35191858) (← links)
- Evidence of a bacterial receptor for lysozyme: binding of lysozyme to the anti-σ factor RsiV controls activation of the ecf σ factor σV. (Q35295202) (← links)
- Clostridium difficile has an original peptidoglycan structure with a high level of N-acetylglucosamine deacetylation and mainly 3-3 cross-links (Q35309863) (← links)
- Wall teichoic acids of Staphylococcus aureus limit recognition by the drosophila peptidoglycan recognition protein-SA to promote pathogenicity. (Q35586976) (← links)
- Diaminopimelic Acid Amidation in Corynebacteriales: NEW INSIGHTS INTO THE ROLE OF LtsA IN PEPTIDOGLYCAN MODIFICATION (Q35595842) (← links)
- Physics of bacterial morphogenesis (Q35598316) (← links)
- Peptidoglycan hydrolases of Escherichia coli (Q35598330) (← links)
- Control of Morphological Differentiation of Streptomyces coelicolor A3(2) by Phosphorylation of MreC and PBP2 (Q35619075) (← links)
- A widespread bacterial type VI secretion effector superfamily identified using a heuristic approach (Q35982125) (← links)
- Clostridium difficile infection: toxins and non-toxin virulence factors, and their contributions to disease establishment and host response (Q36019350) (← links)
- Secondary cell wall polymers of Enterococcus faecalis are critical for resistance to complement activation via mannose-binding lectin (Q36368198) (← links)