Pages that link to "Q36158655"

The following pages link to In the absence of the invariant chain, HLA-DR molecules display a distinct array of peptides which is influenced by the presence or absence of HLA-DM (Q36158655):

Displaying 26 items.

• An MHC class II restriction bias in CD4 T cell responses toward I-A is altered to I-E in DM-deficient mice (Q27469164) (← links)
• Achieving stability through editing and chaperoning: regulation of MHC class II peptide binding and expression (Q28273723) (← links)
• Understanding the mechanism of action of bacterial superantigens from a decade of research. (Q33683420) (← links)
• Interaction of human immunodeficiency virus type 2 Vpx and invariant chain (Q33807088) (← links)
• Peptide exchange in MHC molecules (Q33815435) (← links)
• Accessory molecules for MHC class II peptide loading (Q33840502) (← links)
• Genetic modulation of tumor antigen presentation (Q33877066) (← links)
• HLA-DM and the MHC class II antigen presentation pathway (Q33878174) (← links)
• On the perils of poor editing: regulation of peptide loading by HLA-DQ and H2-A molecules associated with celiac disease and type 1 diabetes (Q34150236) (← links)
• Binding interactions between peptides and proteins of the class II major histocompatibility complex (Q34540061) (← links)
• Dendritic cell maturation and antigen presentation in the absence of invariant chain. (Q35789511) (← links)
• The impact of DM on MHC class II-restricted antigen presentation can be altered by manipulation of MHC-peptide kinetic stability (Q36238108) (← links)
• The relationship between immunodominance, DM editing, and the kinetic stability of MHC class II:peptide complexes (Q36266503) (← links)
• Understanding the focused CD4 T cell response to antigen and pathogenic organisms (Q37386987) (← links)
• Exposing the Specific Roles of the Invariant Chain Isoforms in Shaping the MHC Class II Peptidome (Q37389304) (← links)
• Beyond the classical: influenza virus and the elucidation of alternative MHC class II-restricted antigen processing pathways (Q37958695) (← links)
• Human major histocompatibility molecules have the intrinsic ability to form homotypic associations (Q40877951) (← links)
• HLA-DM can partially replace the invariant chain for HLA-DR transport and surface expression in transfected endocrine epithelial cells (Q40947677) (← links)
• MHC class II presentation of endogenous tumor antigen by cellular vaccines depends on the endocytic pathway but not H2-M. (Q41920366) (← links)
• Conformational alterations during biosynthesis of HLA-DR3 molecules controlled by invariant chain and HLA-DM. (Q43572152) (← links)
• Effects of Prostaglandin F2α and Progesterone on the Ability of Bovine Luteal Cells to Stimulate T Lymphocyte Proliferation1 (Q44425039) (← links)
• Major histocompatibility complex class II-dependent unfolding, transport, and degradation of endogenous proteins (Q46189567) (← links)
• Secondary structure composition and pH-dependent conformational changes of soluble recombinant HLA-DM. (Q46972584) (← links)
• The Ii41 isoform of invariant chain mediates both positive and negative selection events in T-cell receptor transgenic mice. (Q54662060) (← links)
• HLA-DMB expression by thyrocytes: indication of the antigen-processing and possible presenting capability of thyroid cells (Q77344128) (← links)
• Antigen-presenting cells in rheumatoid arthritis (Q77627717) (← links)