Pages that link to "Q35202714"
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The following pages link to Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth (Q35202714):
Displaying 50 items.
- Notch Signaling in Neuroendocrine Tumors (Q26752380) (← links)
- Role of Notch signaling in regulating innate immunity and inflammation in health and disease (Q26764946) (← links)
- Epstein-Barr virus latent genes (Q28082701) (← links)
- The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth (Q28546003) (← links)
- EBNA2 Drives Formation of New Chromosome Binding Sites and Target Genes for B-Cell Master Regulatory Transcription Factors RBP-jκ and EBF1 (Q28552335) (← links)
- Large-scale quality analysis of published ChIP-seq data (Q30717456) (← links)
- BS69/ZMYND11 C-Terminal Domains Bind and Inhibit EBNA2. (Q30981859) (← links)
- Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection (Q33658144) (← links)
- jMOSAiCS: joint analysis of multiple ChIP-seq datasets (Q33741818) (← links)
- The NF-κB genomic landscape in lymphoblastoid B cells (Q34175522) (← links)
- Identification of a recurrent transforming UBR5-ZNF423 fusion gene in EBV-associated nasopharyngeal carcinoma. (Q34200710) (← links)
- Notch inhibitors for cancer treatment (Q34330869) (← links)
- Abortive lytic reactivation of KSHV in CBF1/CSL deficient human B cell lines (Q34733560) (← links)
- Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes (Q34985686) (← links)
- Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming (Q34998243) (← links)
- Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9 (Q34998257) (← links)
- Transcriptomic profile reveals gender-specific molecular mechanisms driving multiple sclerosis progression (Q35109078) (← links)
- RBPJ, the major transcriptional effector of Notch signaling, remains associated with chromatin throughout mitosis, suggesting a role in mitotic bookmarking (Q35112753) (← links)
- Genome-wide analysis reveals conserved and divergent features of Notch1/RBPJ binding in human and murine T-lymphoblastic leukemia cells (Q35202653) (← links)
- Transcription factor RBPJ/CSL: a genome-wide look at transcriptional regulation (Q35202831) (← links)
- EBNA2 binds to genomic intervals associated with multiple sclerosis and overlaps with vitamin D receptor occupancy. (Q35597330) (← links)
- The EBNA-2 N-Terminal Transactivation Domain Folds into a Dimeric Structure Required for Target Gene Activation (Q35664999) (← links)
- EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells (Q35889902) (← links)
- Epstein-Barr virus oncoprotein super-enhancers control B cell growth (Q35963272) (← links)
- The double-edged sword of Notch signaling in cancer (Q35989577) (← links)
- Identification of MEF2B, EBF1, and IL6R as Direct Gene Targets of Epstein-Barr Virus (EBV) Nuclear Antigen 1 Critical for EBV-Infected B-Lymphocyte Survival (Q36433955) (← links)
- A Novel Secreted Protein, MYR1, Is Central to Toxoplasma's Manipulation of Host Cells (Q36548282) (← links)
- Activation of an endothelial Notch1-Jagged1 circuit induces VCAM1 expression, an effect amplified by interleukin-1β. (Q36685371) (← links)
- RBP-J is required for M2 macrophage polarization in response to chitin and mediates expression of a subset of M2 genes (Q36685944) (← links)
- Induction of Epstein-Barr Virus Oncoprotein LMP1 by Transcription Factors AP-2 and Early B Cell Factor (Q36736410) (← links)
- Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites (Q36736629) (← links)
- The B cell transcription program mediates hypomethylation and overexpression of key genes in Epstein-Barr virus-associated proliferative conversion. (Q36872499) (← links)
- MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs. (Q37217649) (← links)
- Epstein-Barr virus nuclear antigen leader protein localizes to promoters and enhancers with cell transcription factors and EBNA2. (Q37319836) (← links)
- Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A. (Q37474974) (← links)
- Epstein-Barr virus super-enhancer eRNAs are essential for MYC oncogene expression and lymphoblast proliferation (Q37493384) (← links)
- Statin-induced changes in gene expression in EBV-transformed and native B-cells (Q37572122) (← links)
- Epigenomic elements enriched in the promoters of autoimmunity susceptibility genes (Q37655858) (← links)
- Epstein-Barr virus EBNA2 directs doxorubicin resistance of B cell lymphoma through CCL3 and CCL4-mediated activation of NF-κB and Btk (Q37706355) (← links)
- Notch signaling and M1 macrophage activation in obesity-alcohol synergism (Q38550320) (← links)
- The RUNX complex: reaching beyond haematopoiesis into immunity (Q38591845) (← links)
- Chromatin Structure of Epstein-Barr Virus Latent Episomes (Q38596337) (← links)
- EBNA2 and Its Coactivator EBNA-LP. (Q38597233) (← links)
- Epstein-Barr virus latency: current and future perspectives (Q38603533) (← links)
- EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency (Q38644507) (← links)
- Epigenetic Alterations in Epstein-Barr Virus-Associated Diseases (Q38667123) (← links)
- Nasopharyngeal carcinoma super-enhancer-driven ETV6 correlates with prognosis (Q38694375) (← links)
- Exploring the mechanisms of genome-wide long-range interactions: interpreting chromosome organization. (Q38697271) (← links)
- Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus Latency (Q38724883) (← links)
- Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression (Q38728190) (← links)