Pages that link to "Q28362068"
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The following pages link to Replicative senescence of biliary epithelial cells precedes bile duct loss in chronic liver allograft rejection: increased expression of p21(WAF1/Cip1) as a disease marker and the influence of immunosuppressive drugs (Q28362068):
Displaying 44 items.
- From immunosuppression to tolerance (Q26822887) (← links)
- Primary biliary cirrhosis is a generalized autoimmune epithelitis (Q27016114) (← links)
- NIH Consensus development project on criteria for clinical trials in chronic graft-versus-host disease: II. The 2014 Pathology Working Group Report (Q28081289) (← links)
- Biliary wound healing, ductular reactions, and IL-6/gp130 signaling in the development of liver disease (Q33866788) (← links)
- Recurrent hepatitis C in liver allografts: prospective assessment of diagnostic accuracy, identification of pitfalls, and observations about pathogenesis (Q34280934) (← links)
- Biliary epithelial apoptosis, autophagy, and senescence in primary biliary cirrhosis (Q34341006) (← links)
- Replicative senescence in organ transplantation-mechanisms and significance (Q34786096) (← links)
- Rejection and recipient age. (Q34823896) (← links)
- Hepatocyte growth factor attenuates liver fibrosis induced by bile duct ligation (Q35088302) (← links)
- Functional role of cellular senescence in biliary injury (Q35143731) (← links)
- Novel approach to bile duct damage in primary biliary cirrhosis: participation of cellular senescence and autophagy (Q35203291) (← links)
- Decreased expression of Bmi1 is closely associated with cellular senescence in small bile ducts in primary biliary cirrhosis (Q35222111) (← links)
- Telomere shortening and cellular senescence in a model of chronic renal allograft rejection (Q35749227) (← links)
- Abnormal Localization of STK17A in Bile Canaliculi in Liver Allografts: An Early Sign of Chronic Rejection (Q35754440) (← links)
- Cholangiocytes with mesenchymal features contribute to progressive hepatic fibrosis of the polycystic kidney rat (Q36202846) (← links)
- Molecular mechanisms of cholangiopathy in primary biliary cirrhosis (Q36527452) (← links)
- Liver biopsy interpretation for causes of late liver allograft dysfunction (Q36548482) (← links)
- Distinguishing between recurrent primary sclerosing cholangitis and chronic rejection (Q36628327) (← links)
- Biliary epithelial senescence and plasticity in acute cellular rejection (Q37101672) (← links)
- The immunopathology of human biliary cell epithelium (Q37521970) (← links)
- Current views on rejection pathology in liver transplantation (Q37781117) (← links)
- New insights on molecular mechanisms of renal aging (Q38033716) (← links)
- Post-transplant liver biopsy and the immune response: lessons for the clinician (Q38053173) (← links)
- Mechanisms of tissue injury in autoimmune liver diseases (Q38235918) (← links)
- Novel therapeutic targets in primary biliary cirrhosis (Q38342107) (← links)
- Etiopathogenesis of primary biliary cirrhosis: an overview of recent developments. (Q38552201) (← links)
- Inhibition of notch signaling pathway prevents cholestatic liver fibrosis by decreasing the differentiation of hepatic progenitor cells into cholangiocytes (Q38809882) (← links)
- Epithelial cell senescence: an adaptive response to pre-carcinogenic stresses? (Q39434311) (← links)
- T cell-mediated biliary epithelial-to-mesenchymal transition in liver allograft rejection. (Q39707815) (← links)
- Chemokine-chemokine receptor CCL2-CCR2 and CX3CL1-CX3CR1 axis may play a role in the aggravated inflammation in primary biliary cirrhosis (Q42258079) (← links)
- Early HCV-associated stellate cell activation in aggressive recurrent HCV: what can liver allografts teach about HCV pathogenesis? (Q42995442) (← links)
- Histologic and biochemical changes during the evolution of chronic rejection of liver allografts (Q43899935) (← links)
- Long-term survival and impact of ursodeoxycholic acid treatment for recurrent primary biliary cirrhosis after liver transplantation (Q44169502) (← links)
- Immunosuppression affects the rate of recurrent primary biliary cirrhosis after liver transplantation (Q44817502) (← links)
- Proinflammatory cytokine-induced cellular senescence of biliary epithelial cells is mediated via oxidative stress and activation of ATM pathway: a culture study (Q46496427) (← links)
- Targeting Senescent Cells in Fibrosis: Pathology, Paradox, and Practical Considerations (Q48522005) (← links)
- Cellular senescence in gastrointestinal diseases: from pathogenesis to therapeutics (Q49565626) (← links)
- Hepatic stellate cell activation in liver transplant patients with hepatitis C recurrence and in non-transplanted patients with chronic hepatitis C. (Q50689772) (← links)
- Huang Qi Decoction Prevents BDL-Induced Liver Fibrosis Through Inhibition of Notch Signaling Activation. (Q51195345) (← links)
- In recurrent primary biliary cirrhosis after liver transplantation, biliary epithelial cells show increased expression of mitochondrial proteins. (Q52925696) (← links)
- An inhibitor of cyclin-dependent kinase, stress-induced p21Waf-1/Cip-1, mediates hepatocyte mito-inhibition during the evolution of cirrhosis. (Q53358254) (← links)
- The Secretin/Secretin Receptor Axis Modulates Ductular Reaction and Liver Fibrosis through Changes in Transforming Growth Factor-β1-Mediated Biliary Senescence (Q57106646) (← links)
- TGF-β induces corneal endothelial senescence via increase of mitochondrial reactive oxygen species in chronic corneal allograft failure (Q60049387) (← links)
- Telomere shortening in the damaged small bile ducts in primary biliary cirrhosis reflects ongoing cellular senescence (Q81413581) (← links)