Pages that link to "Q27760543"
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The following pages link to Tumor suppressor p16INK4A: determination of solution structure and analyses of its interaction with cyclin-dependent kinase 4 (Q27760543):
Displaying 50 items.
- Cloning and characterization of the genes encoding the ankyrin repeat and SOCS box-containing proteins Asb-1, Asb-2, Asb-3 and Asb-4 (Q24290599) (← links)
- Structural basis for inhibition of the cyclin-dependent kinase Cdk6 by the tumour suppressor p16INK4a (Q24316348) (← links)
- Ankyrin repeat and SOCS box 3 (ASB3) mediates ubiquitination and degradation of tumor necrosis factor receptor II (Q24529081) (← links)
- Designed to be stable: crystal structure of a consensus ankyrin repeat protein (Q24550750) (← links)
- Tumor suppressor INK4: refinement of p16INK4A structure and determination of p15INK4B structure by comparative modeling and NMR data (Q24672116) (← links)
- Polymer uncrossing and knotting in protein folding, and their role in minimal folding pathways (Q27321094) (← links)
- Crystal structure of the complex of the cyclin D-dependent kinase Cdk6 bound to the cell-cycle inhibitor p19INK4d (Q27765359) (← links)
- Structure of human cyclin-dependent kinase inhibitor p19INK4d: comparison to known ankyrin-repeat-containing structures and implications for the dysfunction of tumor suppressor p16INK4a (Q27765712) (← links)
- Sequential unfolding of ankyrin repeats in tumor suppressor p16 (Q28202100) (← links)
- Cloning and characterization of diacylglycerol kinase iota splice variants in rat brain (Q28574919) (← links)
- The ankyrin repeat: a diversity of interactions on a common structural framework (Q29616794) (← links)
- Functional evaluation of tumour-specific variants of p16INK4a/CDKN2A: correlation with protein structure information. (Q30322978) (← links)
- Interpreting missense variants: comparing computational methods in human disease genes CDKN2A, MLH1, MSH2, MECP2, and tyrosinase (TYR). (Q30360539) (← links)
- Functional, structural, and genetic evaluation of 20 CDKN2A germ line mutations identified in melanoma-prone families or patients. (Q30375327) (← links)
- Phospho-SXXE/D motif mediated TNF receptor 1-TRADD death domain complex formation for T cell activation and migration. (Q30502703) (← links)
- Designing repeat proteins: well-expressed, soluble and stable proteins from combinatorial libraries of consensus ankyrin repeat proteins. (Q30979207) (← links)
- Biophysical characterization of the free IkappaBalpha ankyrin repeat domain in solution. (Q33204174) (← links)
- Dual coding in alternative reading frames correlates with intrinsic protein disorder. (Q33348728) (← links)
- Disruption of chromosome 11 in canine fibrosarcomas highlights an unusual variability of CDKN2B in dogs (Q33488731) (← links)
- Cyclin-dependent kinases: inhibition and substrate recognition. (Q33801822) (← links)
- Structural basis for the modulation of CDK-dependent/independent activity of cyclin D1. (Q33829852) (← links)
- Cyclin D1 is required for transformation by activated Neu and is induced through an E2F-dependent signaling pathway (Q33884776) (← links)
- Cyclin-dependent kinase inhibitors: novel anticancer agents (Q34075028) (← links)
- The INK4A/ARF locus: role in cell cycle control and apoptosis and implications for glioma growth (Q34283464) (← links)
- A novel p16(INK4A) mutation associated with esophageal squamous cell carcinoma in a high risk population (Q34283555) (← links)
- Combinatorial transcription factors. (Q34477875) (← links)
- Selectivity and potency of cyclin-dependent kinase inhibitors (Q34651981) (← links)
- Utilizing NMR to study the structure of growth-inhibitory proteins (Q35141470) (← links)
- Alterations of the tumor suppressor genes CDKN2A (p16(INK4a)), p14(ARF), CDKN2B (p15(INK4b)), and CDKN2C (p18(INK4c)) in atypical and anaplastic meningiomas (Q35747010) (← links)
- Dynamic Protein Interaction Networks and New Structural Paradigms in Signaling (Q35938545) (← links)
- High prevalence of germline CDKN2A mutations in Slovenian cutaneous malignant melanoma families (Q36147888) (← links)
- Individuals with presumably hereditary uveal melanoma do not harbour germline mutations in the coding regions of either the P16INK4A, P14ARF or cdk4 genes (Q36641299) (← links)
- Familial melanoma-associated mutations in p16 uncouple its tumor-suppressor functions (Q36674934) (← links)
- Assessment of functional effects of unclassified genetic variants (Q37308298) (← links)
- Regulatory mechanisms of tumor suppressor P16(INK4A) and their relevance to cancer (Q37880852) (← links)
- Signaling pathway requirements for induction of senescence by telomere homolog oligonucleotides (Q38334543) (← links)
- Evidence that P12, a specific variant of P16(INK4A), plays a suppressive role in human pancreatic carcinogenesis. (Q39144923) (← links)
- Induced expression of p16(INK4a) inhibits both CDK4- and CDK2-associated kinase activity by reassortment of cyclin-CDK-inhibitor complexes (Q39444837) (← links)
- Classifying variants of CDKN2A using computational and laboratory studies (Q39564985) (← links)
- C-terminal domain of p16(INK4a) is adequate in inducing cell cycle arrest, growth inhibition and CDK4/6 interaction similar to the full length protein in HT-1080 fibrosarcoma cells. (Q39635357) (← links)
- IKKbeta specifically binds to P16 and phosphorylates Ser8 of P16. (Q39740254) (← links)
- Human p16gamma, a novel transcriptional variant of p16(INK4A), coexpresses with p16(INK4A) in cancer cells and inhibits cell-cycle progression. (Q40135907) (← links)
- The tumor suppressor p16(INK4a) prevents cell transformation through inhibition of c-Jun phosphorylation and AP-1 activity (Q40399626) (← links)
- Contributions of conserved TPLH tetrapeptides to the conformational stability of ankyrin repeat proteins. (Q40847451) (← links)
- The study of pH-dependent stability shows that the TPLH-mediated hydrogen-bonding network is important for the conformation and stability of human gankyrin. (Q41862064) (← links)
- Tumor suppressor p16(INK4A)/Cdkn2a alterations in 7, 12-dimethylbenz(a)anthracene (DMBA)-induced hamster cheek pouch tumors (Q41977782) (← links)
- Dissection of protein-protein interaction and CDK4 inhibition in the oncogenic versus tumor suppressing functions of gankyrin and P16. (Q41982097) (← links)
- Simulation of different truncated p16(INK4a) forms and in silico study of interaction with Cdk4. (Q42020862) (← links)
- Cyclin‑dependent kinase inhibitor p21 does not impact embryonic endochondral ossification in mice. (Q42033706) (← links)
- Two arginine rich domains in the p14ARF tumour suppressor mediate nucleolar localization. (Q42628813) (← links)