Pages that link to "Q24311298"

The following pages link to Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death (Q24311298):

Displaying 50 items.

• BCL2 associated agonist of cell death (Q812784) (← links)
• B cell leukemia/lymphoma 2 (Q14863583) (← links)
• BCL2-associated X protein (Q14905404) (← links)
• BCL2-associated agonist of cell death (Q14907709) (← links)
• Vapor of volatile oils from Litsea cubeba seed induces apoptosis and causes cell cycle arrest in lung cancer cells (Q21090858) (← links)
• Analysis of transient phosphorylation-dependent protein-protein interactions in living mammalian cells using split-TEV (Q21256648) (← links)
• Alterations in cell cycle and induction of apoptotic cell death in breast cancer cells treated with α-mangostin extracted from mangosteen pericarp (Q21285004) (← links)
• The phosphorylation-dependent regulation of mitochondrial proteins in stress responses (Q21285141) (← links)
• BCL2-like 1 (Q21497061) (← links)
• Diva, a Bcl-2 homologue that binds directly to Apaf-1 and induces BH3-independent cell death (Q22008056) (← links)
• Identification and characterization of baxepsilon, a novel bax variant missing the BH2 and the transmembrane domains (Q22008718) (← links)
• SLUG, a ces-1-related zinc finger transcription factor gene with antiapoptotic activity, is a downstream target of the E2A-HLF oncoprotein (Q22010638) (← links)
• Rsk1 mediates a MEK-MAP kinase cell survival signal (Q22253213) (← links)
• MCL-1S, a splicing variant of the antiapoptotic BCL-2 family member MCL-1, encodes a proapoptotic protein possessing only the BH3 domain (Q22254196) (← links)
• tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c (Q24290118) (← links)
• MAP-1, a novel proapoptotic protein containing a BH3-like motif that associates with Bax through its Bcl-2 homology domains (Q24290468) (← links)
• The growth-promoting activity of the Bad protein in chicken embryo fibroblasts requires binding to protein 14-3-3 (Q24291620) (← links)
• Functional conservation of 14-3-3 isoforms in inhibiting bad-induced apoptosis (Q24291878) (← links)
• The association of Aiolos transcription factor and Bcl-xL is involved in the control of apoptosis (Q24291916) (← links)
• Proto-oncogene, Pim-3 with serine/threonine kinase activity, is aberrantly expressed in human colon cancer cells and can prevent Bad-mediated apoptosis (Q24295043) (← links)
• Interaction with a membrane surface triggers a reversible conformational change in Bax normally associated with induction of apoptosis (Q24296781) (← links)
• High-throughput methods to detect dimerization of Bcl-2 family proteins (Q24299128) (← links)
• Glioma oncoprotein Bcl2L12 inhibits the p53 tumor suppressor (Q24299628) (← links)
• EGR2 induces apoptosis in various cancer cell lines by direct transactivation of BNIP3L and BAK (Q24299669) (← links)
• Protection from rapamycin-induced apoptosis by insulin-like growth factor-I is partially dependent on protein kinase C signaling (Q24300773) (← links)
• Induction of apoptosis by p110C requires mitochondrial translocation of the proapoptotic BCL-2 family member BAD (Q24301096) (← links)
• Regulation of thymocyte homeostasis by Fliz1 (Q24303449) (← links)
• HSpin1, a transmembrane protein interacting with Bcl-2/Bcl-xL, induces a caspase-independent autophagic cell death (Q24305591) (← links)
• Pore-forming activity of BAD is regulated by specific phosphorylation and structural transitions of the C-terminal part (Q24307486) (← links)
• The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4 (Q24309225) (← links)
• Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis (Q24310597) (← links)
• Dimerization properties of human BAD. Identification of a BH-3 domain and analysis of its binding to mutant BCL-2 and BCL-XL proteins (Q24310631) (← links)
• Expression of the Bcl-2 protein BAD promotes prostate cancer growth (Q24313520) (← links)
• Protein phosphatase 2A dephosphorylation of phosphoserine 112 plays the gatekeeper role for BAD-mediated apoptosis (Q24318719) (← links)
• p28 Bap31, a Bcl-2/Bcl-XL- and procaspase-8-associated protein in the endoplasmic reticulum (Q24318875) (← links)
• Anti-apoptotic actions of vasopressin in H32 neurons involve MAP kinase transactivation and Bad phosphorylation (Q24318888) (← links)
• Identification of novel in vivo phosphorylation sites of the human proapoptotic protein BAD: pore-forming activity of BAD is regulated by phosphorylation (Q24323069) (← links)
• The p53-binding protein 53BP2 also interacts with Bc12 and impedes cell cycle progression at G2/M (Q24323137) (← links)
• The BH3-only protein Bad confers breast cancer taxane sensitivity through a nonapoptotic mechanism (Q24337752) (← links)
• Regulation of BAD by cAMP-dependent protein kinase is mediated via phosphorylation of a novel site, Ser155 (Q24532078) (← links)
• harakiri, a novel regulator of cell death, encodes a protein that activates apoptosis and interacts selectively with survival-promoting proteins Bcl-2 and Bcl-X(L) (Q24532147) (← links)
• Meaningful relationships: the regulation of the Ras/Raf/MEK/ERK pathway by protein interactions (Q24532168) (← links)
• Bim: a novel member of the Bcl-2 family that promotes apoptosis (Q24532974) (← links)
• Association of insulin receptor substrate proteins with Bcl-2 and their effects on its phosphorylation and antiapoptotic function (Q24548364) (← links)
• Multiple Bcl-2 family members demonstrate selective dimerizations with Bax (Q24561496) (← links)
• Protein phosphatase 1alpha is a Ras-activated Bad phosphatase that regulates interleukin-2 deprivation-induced apoptosis (Q24594034) (← links)
• A conserved domain in Bak, distinct from BH1 and BH2, mediates cell death and protein binding functions (Q24597482) (← links)
• TIP30 has an intrinsic kinase activity required for up-regulation of a subset of apoptotic genes (Q24600874) (← links)
• Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis (Q24669892) (← links)
• Rationale for Bcl-xL/Bad peptide complex formation from structure, mutagenesis, and biophysical studies (Q24672334) (← links)