Pages that link to "Q21090537"

The following pages link to Influenza virus PB1-F2 protein induces cell death through mitochondrial ANT3 and VDAC1. (Q21090537):

Displaying 50 items.

• Emerging roles for the influenza A virus nuclear export protein (NEP) (Q21089600) (← links)
• A single mutation in the PB1-F2 of H5N1 (HK/97) and 1918 influenza A viruses contributes to increased virulence (Q21089626) (← links)
• Pathology, molecular biology, and pathogenesis of avian influenza A (H5N1) infection in humans (Q22255463) (← links)
• Antiviral response in pandemic influenza viruses (Q22305645) (← links)
• mTOR/p70S6K signaling distinguishes routine, maintenance-level autophagy from autophagic cell death during influenza A infection (Q24567565) (← links)
• Oncolytic specificity of Newcastle disease virus is mediated by selectivity for apoptosis-resistant cells (Q24602706) (← links)
• Genomic signatures of human versus avian influenza A viruses (Q24623524) (← links)
• Identification of potential conserved RNA secondary structure throughout influenza A coding regions (Q24633659) (← links)
• Genomic analysis of increased host immune and cell death responses induced by 1918 influenza virus (Q24652550) (← links)
• Role of Autophagy and Apoptosis in the Postinfluenza Bacterial Pneumonia (Q26744768) (← links)
• Development of next-generation respiratory virus vaccines through targeted modifications to viral immunomodulatory genes (Q26782411) (← links)
• Norovirus regulation of the innate immune response and apoptosis occurs via the product of the alternative open reading frame 4 (Q27348186) (← links)
• Innate immunity to influenza virus: implications for future therapy (Q27489605) (← links)
• Multi-Omics Studies towards Novel Modulators of Influenza A Virus-Host Interaction (Q28073474) (← links)
• Assessment of the internal genes of influenza A (H7N9) virus contributing to high pathogenicity in mice (Q28386118) (← links)
• The proapoptotic influenza A virus protein PB1-F2 forms a nonselective ion channel (Q28474394) (← links)
• Cellular RNA binding proteins NS1-BP and hnRNP K regulate influenza A virus RNA splicing (Q28534367) (← links)
• Emergence and pandemic potential of swine-origin H1N1 influenza virus (Q29619003) (← links)
• Switch from protective to adverse inflammation during influenza: viral determinants and hemostasis are caught as culprits. (Q30354127) (← links)
• Evolution and adaptation of H5N1 influenza virus in avian and human hosts in Indonesia and Vietnam. (Q30354492) (← links)
• Influenza A virus PB1-F2 protein contributes to viral pathogenesis in mice (Q30355968) (← links)
• Influenza A virus infections in swine: pathogenesis and diagnosis. (Q30357141) (← links)
• The PB1-F2 protein of Influenza A virus: increasing pathogenicity by disrupting alveolar macrophages. (Q30359236) (← links)
• Molecular basis of a pandemic of avian-type influenza virus. (Q30365638) (← links)
• Expression of the 1918 influenza A virus PB1-F2 enhances the pathogenesis of viral and secondary bacterial pneumonia (Q30365855) (← links)
• Survival analysis of infected mice reveals pathogenic variations in the genome of avian H1N1 viruses (Q30369689) (← links)
• Progress in identifying virulence determinants of the 1918 H1N1 and the Southeast Asian H5N1 influenza A viruses. (Q30369896) (← links)
• H5N1 avian influenza virus induces apoptotic cell death in mammalian airway epithelial cells. (Q30372027) (← links)
• Twenty amino acids at the C-terminus of PA-X are associated with increased influenza A virus replication and pathogenicity. (Q30373730) (← links)
• Novel swine-origin influenza A virus in humans: another pandemic knocking at the door. (Q30378285) (← links)
• Use of animal models to understand the pandemic potential of highly pathogenic avian influenza viruses. (Q30379985) (← links)
• H5N1 influenza viruses: outbreaks and biological properties (Q30382229) (← links)
• A Serine12Stop mutation in PB1-F2 of the 2009 pandemic (H1N1) influenza A: a possible reason for its enhanced transmission and pathogenicity to humans. (Q30382800) (← links)
• The Contribution of the PB1-F2 Protein to the Fitness of Influenza A Viruses and its Recent Evolution in the 2009 Influenza A (H1N1) Pandemic Virus. (Q30383785) (← links)
• PB1-F2 expression by the 2009 pandemic H1N1 influenza virus has minimal impact on virulence in animal models (Q30385984) (← links)
• Pandemic influenza A (H1N1) virus infection and avian influenza A (H5N1) virus infection: a comparative analysis (Q30391696) (← links)
• PB1-F2 proteins from H5N1 and 20 century pandemic influenza viruses cause immunopathology (Q30391867) (← links)
• Molecular characterization and comparative analysis of pandemic H1N1/2009 strains with co-circulating seasonal H1N1/2009 strains from eastern India (Q30395362) (← links)
• The 2009 Pandemic H1N1 and Triple-Reassortant Swine H1N1 Influenza Viruses Replicate Efficiently but Elicit an Attenuated Inflammatory Response in Polarized Human Bronchial Epithelial Cells (Q30395821) (← links)
• Virulence determinants of pandemic influenza viruses (Q30398168) (← links)
• Effect of 1918 PB1-F2 expression on influenza A virus infection kinetics (Q30400455) (← links)
• Influenza A viruses: new research developments (Q30404670) (← links)
• The pathogenesis of influenza virus infections: the contributions of virus and host factors (Q30405941) (← links)
• Differential contribution of PB1-F2 to the virulence of highly pathogenic H5N1 influenza A virus in mammalian and avian species (Q30406151) (← links)
• The influenza A virus protein PB1-F2: killing two birds with one stone? (Q30407763) (← links)
• A(H5N1) Virus Evolution in South East Asia (Q30408089) (← links)
• Influenza virus protein PB1-F2 inhibits the induction of type I interferon by binding to MAVS and decreasing mitochondrial membrane potential (Q30417679) (← links)
• Molecular character of influenza A/H1N1 2009: Implications for spread and control (Q30422857) (← links)
• An overview of the highly pathogenic H5N1 influenza virus (Q30426011) (← links)
• H5N1 pathogenesis studies in mammalian models (Q30427806) (← links)