MafB/c-Maf deficiency enables self-renewal of differentiated functional macrophages. (Q34020049)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19892988%20AND%20SRC:MED&resulttype=core&format=json

11 February 2020

title

MafB/c-Maf deficiency enables self-renewal of differentiated functional macrophages (English)

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11 February 2020

1 reference

1 reference

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11 February 2020

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19892988%20AND%20SRC:MED&resulttype=core&format=json

11 February 2020

Athar Aziz

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19892988%20AND%20SRC:MED&resulttype=core&format=json

11 February 2020

Sandrine Sarrazin

series ordinal

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11 February 2020

language of work or name

English

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publication date

1 November 2009

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11 February 2020

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19892988%20AND%20SRC:MED&resulttype=core&format=json

11 February 2020

volume

326

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issue

5954

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11 February 2020

page(s)

867-871

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A double-assurance mechanism controls cell cycle exit upon terminal differentiation in Drosophila.

11 February 2020

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Selective immortalization of murine macrophages from fresh bone marrow by a raf/myc recombinant murine retrovirus

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Local self-renewal can sustain CNS microglia maintenance and function throughout adult life

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MafB is an interaction partner and repressor of Ets-1 that inhibits erythroid differentiation

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