Pages that link to "Q36732878"

The following pages link to DNA damage is a prerequisite for p53-mediated proteasomal degradation of HIF-1alpha in hypoxic cells and downregulation of the hypoxia marker carbonic anhydrase IX. (Q36732878):

Displaying 47 items.

• Pyruvate dehydrogenase kinase as a novel therapeutic target in oncology (Q21129303) (← links)
• Melanoma: from melanocyte to genetic alterations and clinical options (Q21284811) (← links)
• Carbon nanotubes induce malignant transformation and tumorigenesis of human lung epithelial cells (Q23917072) (← links)
• Histone deacetylase inhibitors repress the transactivation potential of hypoxia-inducible factors independently of direct acetylation of HIF-alpha (Q24308024) (← links)
• Recent developments in targeting carbonic anhydrase IX for cancer therapeutics (Q27013023) (← links)
• Mitochondrial activation by inhibition of PDKII suppresses HIF1a signaling and angiogenesis in cancer (Q28267156) (← links)
• Inhibition of hypoxia-inducible factor-1alpha (HIF-1alpha) protein synthesis by DNA damage inducing agents (Q33580209) (← links)
• Complex regulation of the transactivation function of hypoxia-inducible factor-1 alpha by direct interaction with two distinct domains of the CREB-binding protein/p300. (Q33593991) (← links)
• Expression and activity of carbonic anhydrase IX is associated with metabolic dysfunction in MDA-MB-231 breast cancer cells (Q33865240) (← links)
• Involvement of histone deacetylation in MORC2-mediated down-regulation of carbonic anhydrase IX. (Q33871243) (← links)
• Near-infrared fluorescence activation probes based on disassembly-induced emission cyanine dye. (Q33882157) (← links)
• Acrolein-exposed normal human lung fibroblasts in vitro: cellular senescence, enhanced telomere erosion, and degradation of Werner's syndrome protein (Q34131512) (← links)
• Celastrol stimulates hypoxia-inducible factor-1 activity in tumor cells by initiating the ROS/Akt/p70S6K signaling pathway and enhancing hypoxia-inducible factor-1α protein synthesis (Q34482963) (← links)
• Human papillomavirus E7 enhances hypoxia-inducible factor 1-mediated transcription by inhibiting binding of histone deacetylases (Q34799489) (← links)
• Pharmacological activation of a novel p53-dependent S-phase checkpoint involving CHK-1 (Q35064994) (← links)
• Reciprocal influence of the p53 and the hypoxic pathways (Q35065006) (← links)
• p53 activation of mesenchymal stromal cells partially abrogates microenvironment-mediated resistance to FLT3 inhibition in AML through HIF-1α-mediated down-regulation of CXCL12. (Q35485832) (← links)
• Sulforaphane reduces molecular response to hypoxia in ovarian tumor cells independently of their resistance to chemotherapy (Q35798547) (← links)
• HIF-1 and p53: communication of transcription factors under hypoxia. (Q35985516) (← links)
• ING1b negatively regulates HIF1α protein levels in adipose-derived stromal cells by a SUMOylation-dependent mechanism (Q36347178) (← links)
• The p53 codon 72 proline allele is endowed with enhanced cell-death inducing potential in cancer cells exposed to hypoxia. (Q36610337) (← links)
• Inhibition of tumor angiogenesis by p53: a new role for the guardian of the genome. (Q36859052) (← links)
• Regulation of gene expression by hypoxia: integration of the HIF-transduced hypoxic signal at the hypoxia-responsive element (Q36876347) (← links)
• Small-molecule activation of p53 blocks hypoxia-inducible factor 1alpha and vascular endothelial growth factor expression in vivo and leads to tumor cell apoptosis in normoxia and hypoxia (Q37145375) (← links)
• Transcriptional control of the tumor- and hypoxia-marker carbonic anhydrase 9: A one transcription factor (HIF-1) show? (Q37161440) (← links)
• Tumour hypoxia induces a metabolic shift causing acidosis: a common feature in cancer (Q37295258) (← links)
• Loss of PIG3 increases HIF-1α level by promoting protein synthesis via mTOR pathway in renal cell carcinoma cells (Q37317201) (← links)
• Cross-talk between HIF and p53 as mediators of molecular responses to physiological and genotoxic stresses (Q37351778) (← links)
• The quinoxaline di-N-oxide DCQ blocks breast cancer metastasis in vitro and in vivo by targeting the hypoxia inducible factor-1 pathway (Q37599071) (← links)
• Hypoxia inducible factor 1 as a therapeutic target in ischemic stroke (Q37630654) (← links)
• The relevance of protein-protein interactions for p53 function: the CPE contribution. (Q37800755) (← links)
• Carbonic anhydrase IX as an imaging and therapeutic target for tumors and metastases (Q38154554) (← links)
• Metabolic modulation of cancer: a new frontier with great translational potential (Q38317294) (← links)
• Proteasomal inhibition attenuates transcriptional activity of hypoxia-inducible factor 1 (HIF-1) via specific effect on the HIF-1alpha C-terminal activation domain (Q38407792) (← links)
• Expression of HIF-1α ODD domain fused canine caspase 3 by EGFR promoter-driven adenovirus vector induces cytotoxicity in canine breast tumor cells under hypoxia (Q38741055) (← links)
• Synergistic growth inhibition based on small-molecule p53 activation as treatment for intraocular melanoma (Q39505317) (← links)
• Effects of lentivirus-mediated HIF-1alpha knockdown on hypoxia-related cisplatin resistance and their dependence on p53 status in fibrosarcoma cells (Q39990842) (← links)
• Rational design of minimal hypoxia-inducible enhancers (Q39993338) (← links)
• Activation of p53 stimulates proteasome-dependent truncation of eIF4E-binding protein 1 (4E-BP1). (Q40049926) (← links)
• YC-1 inhibits HIF-1 expression in prostate cancer cells: contribution of Akt/NF-kappaB signaling to HIF-1alpha accumulation during hypoxia (Q40184763) (← links)
• The Interaction Between Human Papillomaviruses and the Stromal Microenvironment (Q40444191) (← links)
• In cultured astrocytes, p53 and MDM2 do not alter hypoxia-inducible factor-1alpha function regardless of the presence of DNA damage (Q42616754) (← links)
• A metabolic remodeling in right ventricular hypertrophy is associated with decreased angiogenesis and a transition from a compensated to a decompensated state in pulmonary hypertension (Q46547034) (← links)
• Glucose up-regulates HIF-1 alpha expression in primary cortical neurons in response to hypoxia through maintaining cellular redox status (Q46759642) (← links)
• Testicular hypofunction caused by activating p53 expression induced by reactive oxygen species in varicocele rats (Q46783650) (← links)
• Endogenous hormone 2-methoxyestradiol suppresses venous hypertension-induced angiogenesis through up- and down-regulating p53 and id-1. (Q47386141) (← links)
• p53 mutants cooperate with HIF-1 in transcriptional regulation of extracellular matrix components to promote tumor progression (Q58091439) (← links)