Pages that link to "Q27680878"
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The following pages link to Structural basis of lentiviral subversion of a cellular protein degradation pathway (Q27680878):
Displaying 50 items.
- DDB1 and CUL4 associated factor 1 (Q21112163) (← links)
- New insights into an X-traordinary viral protein (Q21131105) (← links)
- The Expression of Functional Vpx during Pathogenic SIVmac Infections of Rhesus Macaques Suppresses SAMHD1 in CD4+ Memory T Cells (Q27319577) (← links)
- The mechanism of substrate-controlled allosteric regulation of SAMHD1 activated by GTP (Q27698300) (← links)
- The DDB1-DCAF1-Vpr-UNG2 crystal structure reveals how HIV-1 Vpr steers human UNG2 toward destruction (Q27727807) (← links)
- Functional organization of human SAMHD1 and mechanisms of HIV-1 restriction (Q28073284) (← links)
- Interactions with DCAF1 and DDB1 in the CRL4 E3 ubiquitin ligase are required for Vpr-mediated G2 arrest (Q33759415) (← links)
- Interactions between HIV-1 and the cell-autonomous innate immune system. (Q33895123) (← links)
- Nuclear import of SAMHD1 is mediated by a classical karyopherin α/β1 dependent pathway and confers sensitivity to VpxMAC induced ubiquitination and proteasomal degradation (Q33903117) (← links)
- SAMHD1 prevents autoimmunity by maintaining genome stability (Q34399058) (← links)
- Cullin E3 ligases and their rewiring by viral factors (Q34785767) (← links)
- Understanding the molecular manipulation of DCAF1 by the lentiviral accessory proteins Vpr and Vpx. (Q35070909) (← links)
- Molecular determinants for recognition of divergent SAMHD1 proteins by the lentiviral accessory protein Vpx. (Q35450431) (← links)
- Determinants for degradation of SAMHD1, Mus81 and induction of G2 arrest in HIV-1 Vpr and SIVagm Vpr. (Q35684278) (← links)
- Phospho-dependent Regulation of SAMHD1 Oligomerisation Couples Catalysis and Restriction (Q35796953) (← links)
- Structural Basis of Clade-specific Engagement of SAMHD1 (Sterile α Motif and Histidine/Aspartate-containing Protein 1) Restriction Factors by Lentiviral Viral Protein X (Vpx) Virulence Factors (Q35859510) (← links)
- Lineage-Specific Viral Hijacking of Non-canonical E3 Ubiquitin Ligase Cofactors in the Evolution of Vif Anti-APOBEC3 Activity (Q36185332) (← links)
- DCAF1 controls T-cell function via p53-dependent and -independent mechanisms (Q36506967) (← links)
- H11/HSPB8 Restricts HIV-2 Vpx to Restore the Anti-Viral Activity of SAMHD1. (Q36996391) (← links)
- HIV-1 and HIV-2 exhibit divergent interactions with HLTF and UNG2 DNA repair proteins (Q37086412) (← links)
- Expression Profiles of Vpx/Vpr Proteins Are Co-related with the Primate Lentiviral Lineage (Q37146999) (← links)
- Molecular insights into NF2/Merlin tumor suppressor function (Q38204110) (← links)
- The ubiquitin-conjugating system: multiple roles in viral replication and infection (Q38210024) (← links)
- The Multifunctions of WD40 Proteins in Genome Integrity and Cell Cycle Progression (Q38344853) (← links)
- HIV suppression by host restriction factors and viral immune evasion (Q38457723) (← links)
- Inhibition of Vpx-Mediated SAMHD1 and Vpr-Mediated Host Helicase Transcription Factor Degradation by Selective Disruption of Viral CRL4 (DCAF1) E3 Ubiquitin Ligase Assembly (Q38716281) (← links)
- VprBP/DCAF1 Regulates the Degradation and Nonproteolytic Activation of the Cell Cycle Transcription Factor FoxM1. (Q38833602) (← links)
- HIV1-viral protein R (Vpr) mutations: associated phenotypes and relevance for clinical pathologies. (Q38855423) (← links)
- Mutational analysis of HIV-2 Vpx shows that proline residue 109 in the poly-proline motif regulates degradation of SAMHD1. (Q38879647) (← links)
- Degradation of SAMHD1 by Vpx Is Independent of Uncoating (Q38900861) (← links)
- Roles of SAMHD1 in antiviral defense, autoimmunity and cancer. (Q39264008) (← links)
- Cullin-RING E3 Ubiquitin Ligases: Bridges to Destruction. (Q40303257) (← links)
- Vpx overcomes a SAMHD1-independent block to HIV reverse transcription that is specific to resting CD4 T cells. (Q40321547) (← links)
- Characterization of the interactions between SIVrcm Vpx and red-capped mangabey SAMHD1. (Q41407005) (← links)
- HIV: Ringside views (Q42233698) (← links)
- Identification of a conserved interface of HIV-1 and FIV Vifs with Cullin 5. (Q47269109) (← links)
- WD40 repeat domain proteins: a novel target class? (Q47291150) (← links)
- Introduction of H2C2-type zinc-binding residues into HIV-2 Vpr increases its expression level. (Q47555671) (← links)
- A Cyclin-binding Motif in Human SAMHD1 Is Required for Its HIV-1 Restriction, dNTPase Activity, Tetramer Formation, and Efficient Phosphorylation (Q50101944) (← links)
- SAMHD1: Recurring roles in cell cycle, viral restriction, cancer, and innate immunity. (Q52723862) (← links)
- USP18 (UBP43) Abrogates p21-Mediated Inhibition of HIV-1 (Q57469903) (← links)
- Interplay of ancestral non-primate lentiviruses with the virus-restricting SAMHD1 proteins of their hosts (Q58764023) (← links)
- Incomplete Suppression of HIV-1 by SAMHD1 Permits Efficient Macrophage Infection (Q60913086) (← links)
- Cellular Cullin RING Ubiquitin Ligases: Druggable Host Dependency Factors of Cytomegaloviruses (Q63740305) (← links)
- PIM kinases facilitate lentiviral evasion from SAMHD1 restriction via Vpx phosphorylation (Q64097485) (← links)
- HUSH, a Link Between Intrinsic Immunity and HIV Latency (Q64253074) (← links)
- Vpr Targets TET2 for Degradation by CRL4VprBP E3 Ligase to Sustain IL-6 Expression and Enhance HIV-1 Replication (Q89043272) (← links)
- The C-terminal domain of feline and bovine SAMHD1 proteins has a crucial role in lentiviral restriction (Q89790056) (← links)
- HIV-2/SIV Vpx targets a novel functional domain of STING to selectively inhibit cGAS-STING-mediated NF-κB signalling (Q90975678) (← links)
- Conserved Herpesvirus Protein Kinases Target SAMHD1 to Facilitate Virus Replication (Q91770815) (← links)