Pages that link to "Q47950306"
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The following pages link to Expression of apamin receptor in muscles of patients with myotonic muscular dystrophy (Q47950306):
Displaying 42 items.
- Correction of ClC-1 splicing eliminates chloride channelopathy and myotonia in mouse models of myotonic dystrophy (Q24679378) (← links)
- Calcium-activated potassium channels (Q28278153) (← links)
- Single apamin-blocked Ca-activated K+ channels of small conductance in cultured rat skeletal muscle (Q28303268) (← links)
- Identification of a novel protein, DMAP, which interacts with the myotonic dystrophy protein kinase and shows strong homology to D1 snRNP (Q28513178) (← links)
- Presynaptic localization of the small conductance calcium-activated potassium channel SK3 at the neuromuscular junction (Q28569472) (← links)
- Myotonic dystrophy protein kinase is involved in the modulation of the Ca2+ homeostasis in skeletal muscle cells (Q28588083) (← links)
- Equine muscular dystrophy with myotonia (Q31879322) (← links)
- Expression of membrane antigens in myotonic dystrophy (Q33630331) (← links)
- K(+) channels as therapeutic drug targets (Q33960965) (← links)
- Clinical and genetic heterogeneity in myotonic dystrophies (Q34096504) (← links)
- Expanded CUG repeats trigger aberrant splicing of ClC-1 chloride channel pre-mRNA and hyperexcitability of skeletal muscle in myotonic dystrophy (Q34142078) (← links)
- Loss of the muscle-specific chloride channel in type 1 myotonic dystrophy due to misregulated alternative splicing (Q34142085) (← links)
- Increased SK3 expression in DM1 lens cells leads to impaired growth through a greater calcium-induced fragility (Q34581298) (← links)
- Voltage-sensor sodium channel mutations cause hypokalemic periodic paralysis type 2 by enhanced inactivation and reduced current (Q35208537) (← links)
- Myotonia, periodic paralysis and myopathy (Q35229861) (← links)
- Molecular biology of human muscle disease (Q35289330) (← links)
- Muscle chloride channel dysfunction in two mouse models of myotonic dystrophy. (Q36295966) (← links)
- Ion channels and ion transporters of the transverse tubular system of skeletal muscle (Q36577669) (← links)
- Ionic transporting systems of skeletal muscle in relation with innervation and their involvement in myotonic diseases (Q36715356) (← links)
- Sodium and potassium currents in freshly isolated and in proliferating human muscle satellite cells (Q36733237) (← links)
- Chronic muscle stimulation improves muscle function and reverts the abnormal surface EMG pattern in myotonic dystrophy: a pilot study (Q37152085) (← links)
- Calcium-activated potassium channels: regulation by calcium (Q37380235) (← links)
- Myotonic Dystrophy Type 1 Management and Therapeutics (Q39003493) (← links)
- Acute Elevated Glucose Promotes Abnormal Action Potential-Induced Ca2+ Transients in Cultured Skeletal Muscle Fibers (Q41403265) (← links)
- Deficiency of Na+/K(+)-ATPase and sarcoplasmic reticulum Ca(2+)-ATPase in skeletal muscle and cultured muscle cells of myotonic dystrophy patients (Q42181355) (← links)
- Determinants of apamin and d-tubocurarine block in SK potassium channels (Q42547803) (← links)
- Myotonia in colchicine myoneuropathy (Q42551862) (← links)
- Characterization of the high-conductance Ca(2+)-activated K+ channel in adult human skeletal muscle (Q42686719) (← links)
- Abnormalities of the fast sodium current in myotonic dystrophy, recessive generalized myotonia, and adynamia episodica (Q44071015) (← links)
- Inner ear dysfunction in myotonic dystrophy type 1. (Q48310798) (← links)
- Expression of small-conductance calcium-activated potassium channels (SK3) in skeletal muscle: regulation by muscle activity. (Q48479489) (← links)
- Properties of receptors for neurotoxic phospholipases A2 in different tissues (Q48715766) (← links)
- Early subclinical cochlear dysfunction in myotonic dystrophy type 1. (Q50433317) (← links)
- The expression of ion channel mRNAs in skeletal muscles from patients with myotonic muscular dystrophy. (Q52144459) (← links)
- 54th ENMC International Workshop: PROMM (proximal myotonic myopathies) and other proximal myotonic syndromes. 10-12th October 1997, Naarden, The Netherlands. (Q53565802) (← links)
- Properties of Ca2+-activated K+ channels in erythrocytes from patients with myotonic muscular dystrophy. (Q53944704) (← links)
- Voltage-dependent K+ channels in the sarcolemma of mouse skeletal muscle (Q67829630) (← links)
- [Anesthesia in myotonia] (Q69316233) (← links)
- Neural control of the expression of a Ca(2+)-activated K+ channel involved in the induction of myotonic-like characteristics (Q71276424) (← links)
- Decreased expression of myotonic dystrophy protein kinase and disorganization of sarcoplasmic reticulum in skeletal muscle of myotonic dystrophy (Q74579021) (← links)
- A surface EMG analysis of sarcolemma excitability alteration and myofibre degeneration in Steinert disease (Q74608414) (← links)
- UCL 1684: a potent blocker of Ca2+ -activated K+ channels in rat adrenal chromaffin cells in culture (Q74651654) (← links)