Pages that link to "Q39808498"
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The following pages link to Repair of clustered uracil DNA damages in Escherichia coli (Q39808498):
Displaying 32 items.
- Processing of a complex multiply damaged DNA site by human cell extracts and purified repair proteins (Q24793953) (← links)
- Uracil DNA N-glycosylase promotes assembly of human centromere protein A (Q27351194) (← links)
- Apex1 can cleave complex clustered DNA lesions in cells (Q33569273) (← links)
- Saccharomyces cerevisiae-based system for studying clustered DNA damages (Q34005933) (← links)
- Clustered DNA lesion repair in eukaryotes: relevance to mutagenesis and cell survival (Q35004467) (← links)
- Delayed repair of radiation induced clustered DNA damage: friend or foe? (Q35038786) (← links)
- Disruption of PARP1 function inhibits base excision repair of a sub-set of DNA lesions (Q35587953) (← links)
- Replication forks stalled at ultraviolet lesions are rescued via RecA and RuvABC protein-catalyzed disintegration in Escherichia coli (Q35839032) (← links)
- Interplay between DNA N-glycosylases/AP lyases at multiply damaged sites and biological consequences (Q35865301) (← links)
- Chromosome demise in the wake of ligase-deficient replication (Q36016450) (← links)
- Base excision repair processing of abasic site/single-strand break lesions within clustered damage sites associated with XRCC1 deficiency (Q36360631) (← links)
- Human abasic endonuclease action on multilesion abasic clusters: implications for radiation-induced biological damage (Q36649692) (← links)
- Artemis is required to improve the accuracy of repair of double-strand breaks with 5'-blocked termini generated from non-DSB-clustered lesions (Q36778971) (← links)
- DNA repair of clustered lesions in mammalian cells: involvement of non-homologous end-joining (Q36859276) (← links)
- The formation of double-strand breaks at multiply damaged sites is driven by the kinetics of excision/incision at base damage in eukaryotic cells (Q37149749) (← links)
- Replication fork collapse is a major cause of the high mutation frequency at three-base lesion clusters (Q37271098) (← links)
- The yield, processing, and biological consequences of clustered DNA damage induced by ionizing radiation (Q37393656) (← links)
- Closely opposed apurinic/apyrimidinic sites are converted to double strand breaks in Escherichia coli even in the absence of exonuclease III, endonuclease IV, nucleotide excision repair and AP lyase cleavage (Q38317916) (← links)
- Increased mutability and decreased repairability of a three-lesion clustered DNA-damaged site comprised of an AP site and bi-stranded 8-oxoG lesions (Q39018442) (← links)
- Hierarchy of lesion processing governs the repair, double-strand break formation and mutability of three-lesion clustered DNA damage (Q39373211) (← links)
- Participation of DNA-PKcs in DSB Repair after Exposure to High- and Low-LET Radiation (Q39672164) (← links)
- Two clustered 8-oxo-7,8-dihydroguanine (8-oxodG) lesions increase the point mutation frequency of 8-oxodG, but do not result in double strand breaks or deletions in Escherichia coli (Q40405003) (← links)
- Enhanced mutagenic potential of 8-oxo-7,8-dihydroguanine when present within a clustered DNA damage site (Q40695070) (← links)
- Processing of clustered DNA damage generates additional double-strand breaks in mammalian cells post-irradiation (Q40773113) (← links)
- Processing of thymine glycol in a clustered DNA damage site: mutagenic or cytotoxic (Q41825528) (← links)
- The roles of specific glycosylases in determining the mutagenic consequences of clustered DNA base damage (Q41862287) (← links)
- Efficient processing of abasic sites by bacterial nonhomologous end-joining Ku proteins (Q41914861) (← links)
- Stress and survival of aging Escherichia coli rpoS colonies (Q42922063) (← links)
- Evaluation of lesion clustering in irradiated plasmid DNA. (Q46552413) (← links)
- Patterns of chromosomal fragmentation due to uracil-DNA incorporation reveal a novel mechanism of replication-dependent double-stranded breaks (Q46724332) (← links)
- Effects of radiation quality on interactions between oxidative stress, protein and DNA damage in Deinococcus radiodurans. (Q51686478) (← links)
- SSB as an organizer/mobilizer of genome maintenance complexes (Q79661973) (← links)