Pages that link to "Q38359264"
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The following pages link to Reactivation of lytic replication from B cells latently infected with Epstein-Barr virus occurs with high S-phase cyclin-dependent kinase activity while inhibiting cellular DNA replication. (Q38359264):
Displaying 50 items.
- TORC2, a coactivator of cAMP-response element-binding protein, promotes Epstein-Barr virus reactivation from latency through interaction with viral BZLF1 protein (Q24320081) (← links)
- The Epstein-Barr virus replication protein BBLF2/3 provides an origin-tethering function through interaction with the zinc finger DNA binding protein ZBRK1 and the KAP-1 corepressor (Q24558692) (← links)
- Human complement receptor type 1/CD35 is an Epstein-Barr Virus receptor (Q24632150) (← links)
- Preliminary evidence of mitochondrial dysfunction associated with post-infective fatigue after acute infection with Epstein Barr virus (Q25257251) (← links)
- Molecular Mechanisms of White Spot Syndrome Virus Infection and Perspectives on Treatments (Q26770085) (← links)
- Phosphoproteomic analyses reveal signaling pathways that facilitate lytic gammaherpesvirus replication (Q27333727) (← links)
- Classic swine fever virus NS2 protein leads to the induction of cell cycle arrest at S-phase and endoplasmic reticulum stress (Q27490910) (← links)
- Cyclin-dependent kinase-like function is shared by the beta- and gamma- subset of the conserved herpesvirus protein kinases (Q28475457) (← links)
- In vivo dynamics of EBNA1-oriP interaction during latent and lytic replication of Epstein-Barr virus (Q31122638) (← links)
- Homologous recombinational repair factors are recruited and loaded onto the viral DNA genome in Epstein-Barr virus replication compartments (Q33433812) (← links)
- Degradation of phosphorylated p53 by viral protein-ECS E3 ligase complex (Q33489621) (← links)
- Architecture of replication compartments formed during Epstein-Barr virus lytic replication (Q33737678) (← links)
- Hyperphosphorylation of EBNA2 by Epstein-Barr virus protein kinase suppresses transactivation of the LMP1 promoter (Q33754919) (← links)
- Noise cancellation: viral fine tuning of the cellular environment for its own genome replication. (Q33780955) (← links)
- Increased efficiency of phorbol ester-induced lytic reactivation of Kaposi's sarcoma-associated herpesvirus during S phase (Q33781299) (← links)
- Epstein-Barr virus (EBV) Rta-mediated EBV and Kaposi's sarcoma-associated herpesvirus lytic reactivations in 293 cells. (Q33851707) (← links)
- Epstein-Barr virus BGLF4 kinase retards cellular S-phase progression and induces chromosomal abnormality (Q34328083) (← links)
- Inhibition of S-phase cyclin-dependent kinase activity blocks expression of Epstein-Barr virus immediate-early and early genes, preventing viral lytic replication (Q34553293) (← links)
- Identification of herpesvirus proteins that contribute to G1/S arrest (Q35087201) (← links)
- Phosphorylation of MCM4 at sites inactivating DNA helicase activity of the MCM4-MCM6-MCM7 complex during Epstein-Barr virus productive replication (Q35101480) (← links)
- The lytic phase of epstein-barr virus requires a viral genome with 5-methylcytosine residues in CpG sites (Q35666071) (← links)
- Quercetin-induced apoptosis prevents EBV infection (Q35828636) (← links)
- Epstein-Barr virus BGLF4 kinase induces premature chromosome condensation through activation of condensin and topoisomerase II. (Q35857467) (← links)
- Deregulation of the cell cycle by the Epstein-Barr virus (Q35941628) (← links)
- EBV and not HPV sensitizes tobacco-associated head and neck cancer cell line FaDu to radiotherapy (Q36772369) (← links)
- Regulation of the retinoblastoma proteins by the human herpesviruses. (Q37085853) (← links)
- White spot syndrome virus IE1 and WSV056 modulate the G1/S transition by binding to the host retinoblastoma protein (Q37336538) (← links)
- Identification of a negative regulatory element in the Epstein-Barr virus Zta transactivation domain that is regulated by the cell cycle control factors c-Myc and E2F1. (Q37583477) (← links)
- Epigenetic modification of the Epstein-Barr virus BZLF1 promoter regulates viral reactivation from latency (Q38098179) (← links)
- Switching of EBV cycles between latent and lytic states (Q38171455) (← links)
- Regulation of Epstein-Barr virus reactivation from latency (Q38208532) (← links)
- Studying classical swine fever virus: making the best of a bad virus (Q38289135) (← links)
- The long-lasting love affair between the budding yeast Saccharomyces cerevisiae and the Epstein-Barr virus (Q38574687) (← links)
- An epidemiological and molecular study of the relationship between smoking, risk of nasopharyngeal carcinoma, and Epstein-Barr virus activation (Q39279923) (← links)
- Epigenetic histone modification of Epstein-Barr virus BZLF1 promoter during latency and reactivation in Raji cells (Q39392105) (← links)
- Nuclear transport of Epstein-Barr virus DNA polymerase is dependent on the BMRF1 polymerase processivity factor and molecular chaperone Hsp90 (Q39440772) (← links)
- Spatiotemporally different DNA repair systems participate in Epstein-Barr virus genome maturation (Q39559227) (← links)
- Epstein-Barr virus deubiquitinase downregulates TRAF6-mediated NF-κB signaling during productive replication. (Q39789708) (← links)
- Efficient production of infectious viruses requires enzymatic activity of Epstein-Barr virus protein kinase. (Q39854651) (← links)
- Expression of Epstein-Barr virus BZLF1 immediate-early protein induces p53 degradation independent of MDM2, leading to repression of p53-mediated transcription. (Q39859670) (← links)
- Plasma cell-specific transcription factor XBP-1s binds to and transactivates the Epstein-Barr virus BZLF1 promoter (Q40075775) (← links)
- The Translesion Polymerase Pol η Is Required for Efficient Epstein-Barr Virus Infectivity and Is Regulated by the Viral Deubiquitinating Enzyme BPLF1. (Q40116028) (← links)
- Epstein-Barr Virus Rta-Mediated Accumulation of DNA Methylation Interferes with CTCF Binding in both Host and Viral Genomes. (Q40207035) (← links)
- Postreplicative mismatch repair factors are recruited to Epstein-Barr virus replication compartments (Q40309902) (← links)
- HCV NS2 protein inhibits cell proliferation and induces cell cycle arrest in the S-phase in mammalian cells through down-regulation of cyclin A expression (Q40312909) (← links)
- The nucleocapsid protein of severe acute respiratory syndrome-coronavirus inhibits the activity of cyclin-cyclin-dependent kinase complex and blocks S phase progression in mammalian cells (Q40326049) (← links)
- KSHV G protein-coupled receptor inhibits lytic gene transcription in primary-effusion lymphoma cells via p21-mediated inhibition of Cdk2. (Q40375800) (← links)
- Activation of ataxia telangiectasia-mutated DNA damage checkpoint signal transduction elicited by herpes simplex virus infection (Q40407545) (← links)
- Epstein-Barr virus lytic replication elicits ATM checkpoint signal transduction while providing an S-phase-like cellular environment. (Q40478598) (← links)
- Cooperation between different forms of the human papillomavirus type 1 E4 protein to block cell cycle progression and cellular DNA synthesis. (Q40487876) (← links)