Pages that link to "Q37712557"
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The following pages link to Integrin-modulating therapy prevents fibrosis and autoimmunity in mouse models of scleroderma (Q37712557):
Displaying 50 items.
- Structural basis for pure antagonism of integrin αVβ3 by a high-affinity form of fibronectin (Q24292863) (← links)
- The clearance of dying cells: table for two (Q26753825) (← links)
- Mechanotransduction and extracellular matrix homeostasis (Q26998075) (← links)
- Pathophysiology of systemic sclerosis (Q28072568) (← links)
- αv integrins: key regulators of tissue fibrosis (Q28076205) (← links)
- Recent advances in the genetics of systemic sclerosis: toward biological and clinical significance (Q28088499) (← links)
- Animal models of systemic sclerosis: their utility and limitations (Q28390528) (← links)
- Prestress in the extracellular matrix sensitizes latent TGF-β1 for activation. (Q30596295) (← links)
- Cellular mechanisms of tissue fibrosis. 8. Current and future drug targets in fibrosis: focus on Rho GTPase-regulated gene transcription. (Q33840143) (← links)
- Pivotal Functions of Plasmacytoid Dendritic Cells in Systemic Autoimmune Pathogenesis (Q33956662) (← links)
- Altered MCM protein levels and autophagic flux in aged and systemic sclerosis dermal fibroblasts (Q34000514) (← links)
- Fibrillin microfibrils in bone physiology (Q34495815) (← links)
- Systems level analysis of systemic sclerosis shows a network of immune and profibrotic pathways connected with genetic polymorphisms. (Q34888501) (← links)
- mTOR inhibition rescues osteopenia in mice with systemic sclerosis (Q34924612) (← links)
- Experimentally-derived fibroblast gene signatures identify molecular pathways associated with distinct subsets of systemic sclerosis patients in three independent cohorts. (Q34995163) (← links)
- Activation of non-canonical TGF-β1 signaling indicates an autoimmune mechanism for bone marrow fibrosis in primary myelofibrosis (Q35113804) (← links)
- Multiplex cytokine analysis of dermal interstitial blister fluid defines local disease mechanisms in systemic sclerosis (Q35545185) (← links)
- Myofibroblasts in murine cutaneous fibrosis originate from adiponectin-positive intradermal progenitors (Q35755610) (← links)
- Fresolimumab treatment decreases biomarkers and improves clinical symptoms in systemic sclerosis patients (Q36040200) (← links)
- The matrix protein Fibulin-5 is at the interface of tissue stiffness and inflammation in fibrosis (Q36251455) (← links)
- Origin of fibrosing cells in systemic sclerosis (Q36264658) (← links)
- Proteinase 3 on apoptotic cells disrupts immune silencing in autoimmune vasculitis (Q36266550) (← links)
- Fibrillin-containing microfibrils are key signal relay stations for cell function (Q36470965) (← links)
- Update on macrophages and innate immunity in scleroderma (Q36607887) (← links)
- Rational design of a protein that binds integrin αvβ3 outside the ligand binding site (Q36974007) (← links)
- ADAMTS-10 and -6 differentially regulate cell-cell junctions and focal adhesions (Q37363956) (← links)
- Dendritic cells maintain dermal adipose-derived stromal cells in skin fibrosis (Q37395851) (← links)
- αvβ1 integrin as a novel therapeutic target for tissue fibrosis (Q37410984) (← links)
- A novel multi-network approach reveals tissue-specific cellular modulators of fibrosis in systemic sclerosis. (Q37717578) (← links)
- Cardiovascular manifestations in Marfan syndrome and related diseases; multiple genes causing similar phenotypes (Q38215407) (← links)
- Molecular and cellular basis of scleroderma (Q38230276) (← links)
- Transforming growth factor β--at the centre of systemic sclerosis (Q38241227) (← links)
- Meaningful prevention of breast cancer metastasis: candidate therapeutics, preclinical validation, and clinical trial concerns (Q38269966) (← links)
- Early systemic sclerosis-opportunities for treatment. (Q38364855) (← links)
- Matrix remodeling in systemic sclerosis (Q38542590) (← links)
- The role of the acquired immune response in systemic sclerosis (Q38543690) (← links)
- A healthy tension in translational research (Q38592372) (← links)
- Critical Appraisal of the Utility and Limitations of Animal Models of Scleroderma (Q38681751) (← links)
- Unifying mechanism for different fibrotic diseases (Q38829704) (← links)
- FBN1: The disease-causing gene for Marfan syndrome and other genetic disorders (Q38903109) (← links)
- Review: Frontiers of Antifibrotic Therapy in Systemic Sclerosis (Q38956376) (← links)
- TGF-β Family Signaling in Connective Tissue and Skeletal Diseases (Q39155183) (← links)
- A microfibril assembly assay identifies different mechanisms of dominance underlying Marfan syndrome, stiff skin syndrome and acromelic dysplasias (Q41373533) (← links)
- Pathophysiological Mechanisms in Sclerosing Skin Diseases (Q41462744) (← links)
- Abnormal mechanosensing and cofilin activation promote the progression of ascending aortic aneurysms in mice (Q41525208) (← links)
- Fell-Muir Lecture: Fibrillin microfibrils: structural tensometers of elastic tissues? (Q42376279) (← links)
- Early fibrillin-1 assembly monitored through a modifiable recombinant cell approach (Q42402963) (← links)
- The RNA-binding protein, ZC3H14, is required for proper poly(A) tail length control, expression of synaptic proteins, and brain function in mice. (Q48217717) (← links)
- T-cell regulation through a basic suppressive mechanism targeting low-density lipoprotein receptor-related protein 1. (Q48330352) (← links)
- Preclinical rationale for TGF-β inhibition as a therapeutic target for the treatment of myelofibrosis. (Q49196561) (← links)