Pages that link to "Q37077297"
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The following pages link to VASCULAR INFLAMMATION AND ATHEROGENESIS ARE ACTIVATED VIA RECEPTORS FOR PAMPs AND SUPPRESSED BY REGULATORY T CELLS. (Q37077297):
Displaying 37 items.
- Interleukin-35 Inhibits Endothelial Cell Activation by Suppressing MAPK-AP-1 Pathway. (Q27312371) (← links)
- Inflammasomes are differentially expressed in cardiovascular and other tissues (Q33762674) (← links)
- The FOX transcription factors regulate vascular pathology, diabetes and Tregs (Q33763033) (← links)
- Inflammatory and autoimmune reactions in atherosclerosis and vaccine design informatics (Q33805176) (← links)
- Th 17 cells interplay with Foxp3+ Tregs in regulation of inflammation and autoimmunity (Q33894387) (← links)
- Immunosuppressive/anti-inflammatory cytokines directly and indirectly inhibit endothelial dysfunction--a novel mechanism for maintaining vascular function (Q34538722) (← links)
- Structural evidence of anti-atherogenic microRNAs (Q35017677) (← links)
- Early hyperlipidemia promotes endothelial activation via a caspase-1-sirtuin 1 pathway (Q35224401) (← links)
- Endothelial progenitor cells in cardiovascular diseases: from biomarker to therapeutic agent (Q35604291) (← links)
- Inhibition of Caspase-1 Activation in Endothelial Cells Improves Angiogenesis: A NOVEL THERAPEUTIC POTENTIAL FOR ISCHEMIA (Q35837196) (← links)
- Endothelial progenitor cells in atherosclerosis (Q36011477) (← links)
- Vasoregression: A Shared Vascular Pathology Underlying Macrovascular And Microvascular Pathologies? (Q36384149) (← links)
- Metabolic Diseases Downregulate the Majority of Histone Modification Enzymes, Making a Few Upregulated Enzymes Novel Therapeutic Targets--"Sand Out and Gold Stays". (Q36619589) (← links)
- Interleukin-17A Promotes Aortic Endothelial Cell Activation via Transcriptionally and Post-translationally Activating p38 Mitogen-activated Protein Kinase (MAPK) Pathway (Q36650270) (← links)
- Identification of novel pretranslational regulatory mechanisms for NF-κB activation. (Q36890591) (← links)
- The Role of Immunogenicity in Cardiovascular Disease (Q37560012) (← links)
- Mitochondrial ROS, uncoupled from ATP synthesis, determine endothelial activation for both physiological recruitment of patrolling cells and pathological recruitment of inflammatory cells (Q37679976) (← links)
- Thrombus leukocytes exhibit more endothelial cell-specific angiogenic markers than peripheral blood leukocytes do in acute coronary syndrome patients, suggesting a possibility of trans-differentiation: a comprehensive database mining study (Q37719815) (← links)
- Coexistence of vasculitides with familial Mediterranean fever (Q37872631) (← links)
- Pathological conditions re-shape physiological Tregs into pathological Tregs (Q38654764) (← links)
- Lysophospholipids and Their Receptors Serve as Conditional DAMPs and DAMP Receptors in Tissue Oxidative and Inflammatory Injury (Q38742072) (← links)
- Nucleic Acid Delivery for Endothelial Dysfunction in Cardiovascular Diseases (Q39003359) (← links)
- IL-35, as a newly proposed homeostasis-associated molecular pattern, plays three major functions including anti-inflammatory initiator, effector, and blocker in cardiovascular diseases. (Q39396961) (← links)
- Metabolism-associated danger signal-induced immune response and reverse immune checkpoint-activated CD40+ monocyte differentiation (Q39454807) (← links)
- Lysophospholipid Receptors, as Novel Conditional Danger Receptors and Homeostatic Receptors Modulate Inflammation-Novel Paradigm and Therapeutic Potential (Q39732801) (← links)
- A comprehensive data mining study shows that most nuclear receptors act as newly proposed homeostasis-associated molecular pattern receptors (Q42634180) (← links)
- Low-Intensity Ultrasound-Induced Anti-inflammatory Effects Are Mediated by Several New Mechanisms Including Gene Induction, Immunosuppressor Cell Promotion, and Enhancement of Exosome Biogenesis and Docking (Q46645885) (← links)
- GATA3, HDAC6, and BCL6 Regulate FOXP3+ Treg Plasticity and Determine Treg Conversion into Either Novel Antigen-Presenting Cell-Like Treg or Th1-Treg (Q49474705) (← links)
- Uremic toxins are conditional danger- or homeostasis-associated molecular patterns (Q49992231) (← links)
- DNA Checkpoint and Repair Factors Are Nuclear Sensors for Intracellular Organelle Stresses-Inflammations and Cancers Can Have High Genomic Risks. (Q55231975) (← links)
- Damage-associated molecular patterns in inflammatory bowel disease: From biomarkers to therapeutic targets (Q58699651) (← links)
- HSP22 suppresses diabetes-induced endothelial injury by inhibiting mitochondrial reactive oxygen species formation (Q60922054) (← links)
- Dynamic Profile of CD4 T-Cell-Associated Cytokines/Chemokines following Murine Myocardial Infarction/Reperfusion (Q64081292) (← links)
- Increased acetylation of H3K14 in the genomic regions that encode trained immunity enzymes in lysophosphatidylcholine-activated human aortic endothelial cells - Novel qualification markers for chronic disease risk factors and conditional DAMPs. (Q64986016) (← links)
- Proton leak regulates mitochondrial reactive oxygen species generation in endothelial cell activation and inflammation - A novel concept (Q90308514) (← links)
- Co-signaling receptors regulate T-cell plasticity and immune tolerance (Q93368983) (← links)
- ROS systems are a new integrated network for sensing homeostasis and alarming stresses in organelle metabolic processes (Q99575245) (← links)