Pages that link to "Q36835203"
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The following pages link to Whole genome scanning as a cytogenetic tool in hematologic malignancies (Q36835203):
Displaying 50 items.
- Institutional shared resources and translational cancer research (Q24288943) (← links)
- FISH and SNP-A karyotyping in myelodysplastic syndromes: improving cytogenetic detection of del(5q), monosomy 7, del(7q), trisomy 8 and del(20q) (Q24644421) (← links)
- Molecular Testing in Myelodysplastic Syndromes for the Practicing Oncologist: Will the Progress Fulfill the Promise? (Q26801573) (← links)
- SNPs Array Karyotyping in Non-Hodgkin Lymphoma (Q28085444) (← links)
- Use of single nucleotide polymorphism array technology to improve the identification of chromosomal lesions in leukemia (Q28390558) (← links)
- Mutations of an E3 ubiquitin ligase c-Cbl but not TET2 mutations are pathogenic in juvenile myelomonocytic leukemia (Q33722741) (← links)
- Copy neutral loss of heterozygosity: a novel chromosomal lesion in myeloid malignancies (Q33788643) (← links)
- The challenging world of cytopenias: distinguishing myelodysplastic syndromes from other disorders of marrow failure (Q33829778) (← links)
- SNPs array karyotyping reveals a novel recurrent 20p13 amplification in primary myelofibrosis (Q34081581) (← links)
- Genome-Wide Mapping Of Copy Number Variations using SNP Arrays (Q34139304) (← links)
- The Biology and Targeting of FLT3 in Pediatric Leukemia (Q34229555) (← links)
- Assessing karyotype precision by microarray-based comparative genomic hybridization in the myelodysplastic/myeloproliferative syndromes (Q34400180) (← links)
- Mutations of e3 ubiquitin ligase cbl family members constitute a novel common pathogenic lesion in myeloid malignancies. (Q34578265) (← links)
- CBL, CBLB, TET2, ASXL1, and IDH1/2 mutations and additional chromosomal aberrations constitute molecular events in chronic myelogenous leukemia. (Q35030952) (← links)
- Loss of the wild-type allele contributes to myeloid expansion and disease aggressiveness in FLT3/ITD knockin mice (Q35525611) (← links)
- Cytogenetic and molecular predictors of response in patients with myeloid malignancies without del[5q] treated with lenalidomide. (Q35881223) (← links)
- Genomic evolution in Barrett's adenocarcinoma cells: critical roles of elevated hsRAD51, homologous recombination and Alu sequences in the genome (Q36120783) (← links)
- Statistical genetic issues for genome-wide association studies (Q36391069) (← links)
- Loss of heterozygosity 4q24 and TET2 mutations associated with myelodysplastic/myeloproliferative neoplasms (Q37262428) (← links)
- Diagnosis and treatment of primary myelodysplastic syndromes in adults: recommendations from the European LeukemiaNet (Q37263146) (← links)
- Single nucleotide polymorphism arrays: a decade of biological, computational and technological advances (Q37273694) (← links)
- Polymorphisms in microRNA-related genes are associated with survival of patients with T-cell lymphoma (Q37644547) (← links)
- Selected technologies for measuring acquired genetic damage in humans (Q37792988) (← links)
- Gene expression profiling in MDS and AML: potential and future avenues. (Q37858877) (← links)
- Pathogenesis and consequences of uniparental disomy in cancer (Q37868831) (← links)
- The Third Consensus Conference on the treatment of aplastic anemia (Q37880645) (← links)
- Optimizing management of myelodysplastic syndromes post-allogeneic transplantation (Q37955235) (← links)
- Molecular diagnosis of leukemia (Q38018998) (← links)
- Array comparative genomic hybridization in pediatric acute leukemias. (Q38113815) (← links)
- Recent advances in understanding the molecular pathogenesis of myelodysplastic syndromes (Q38122703) (← links)
- I walk the line: how to tell MDS from other bone marrow failure conditions (Q38235622) (← links)
- Single-nucleotide polymorphism-array improves detection rate of genomic alterations in core-binding factor leukemia. (Q38315894) (← links)
- A genome-wide single-nucleotide polymorphism-array can improve the prognostic stratification of the core binding factor acute myeloid leukemia. (Q38459577) (← links)
- Genome-wide high density single-nucleotide polymorphism array-based karyotyping improves detection of clonal aberrations including der(9) deletion, but does not predict treatment outcomes after imatinib therapy in chronic myeloid leukemia (Q38476431) (← links)
- Evaluation of chronic lymphocytic leukemia by oligonucleotide-based microarray analysis uncovers novel aberrations not detected by FISH or cytogenetic analysis (Q39686496) (← links)
- What Is the Clinical Utility of Repeat SNP Array Testing in the Follow-up of Myeloid Neoplasms?: A Retrospective Analysis of 44 Patients With Serial SNP Arrays (Q40250091) (← links)
- The vanguard has arrived in the clinical laboratory: array-based karyotyping for prognostic markers in chronic lymphocytic leukemia (Q40609808) (← links)
- TP53 mutations in myeloid malignancies are either homozygous or hemizygous due to copy number-neutral loss of heterozygosity or deletion of 17p. (Q41813518) (← links)
- Cryptic chromosome abnormalities in a patient with mixed phenotype acute leukemia (Q41924078) (← links)
- 250K single nucleotide polymorphism array karyotyping identifies acquired uniparental disomy and homozygous mutations, including novel missense substitutions of c-Cbl, in myeloid malignancies (Q43078973) (← links)
- Metaphase cytogenetics and single nucleotide polymorphism arrays in myeloid malignancies. (Q43212949) (← links)
- Clonal hematopoiesis in Philadelphia chromosome-negative bone marrow cells of chronic myeloid leukemia patients receiving dasatinib. (Q43266877) (← links)
- Microarray-based genomic profiling as a diagnostic tool in acute lymphoblastic leukemia (Q43488736) (← links)
- Identification of acquired copy number alterations and uniparental disomies in cytogenetically normal acute myeloid leukemia using high-resolution single-nucleotide polymorphism analysis (Q44336225) (← links)
- Next-generation sequencing of the TET2 gene in 355 MDS and CMML patients reveals low-abundance mutant clones with early origins, but indicates no definite prognostic value (Q45088115) (← links)
- Genomic, immunophenotypic, and NPM1/FLT3 mutational studies on 17 patients with normal karyotype acute myeloid leukemia (AML) followed by aberrant karyotype AML at relapse (Q51750504) (← links)
- CBL mutation-related patterns of phosphorylation and sensitivity to tyrosine kinase inhibitors. (Q54329349) (← links)
- Additional chromosomal abnormalities detected by array comparative genomic hybridization in AML. (Q54337186) (← links)
- Characterization of chromosome arm 20q abnormalities in myeloid malignancies using genome-wide single nucleotide polymorphism array analysis (Q82616592) (← links)
- Deletions of Xp22.2 including PIG-A locus lead to paroxysmal nocturnal hemoglobinuria (Q82660130) (← links)