Pages that link to "Q34658141"

The following pages link to Depletion of SMN by RNA interference in HeLa cells induces defects in Cajal body formation. (Q34658141):

Displaying 36 items.

• WRAP53 is essential for Cajal body formation and for targeting the survival of motor neuron complex to Cajal bodies (Q21145787) (← links)
• Two distinct arginine methyltransferases are required for biogenesis of Sm-class ribonucleoproteins (Q24337867) (← links)
• Cajal-body formation correlates with differential coilin phosphorylation in primary and transformed cell lines (Q24658020) (← links)
• Dephosphorylation of survival motor neurons (SMN) by PPM1G/PP2Cgamma governs Cajal body localization and stability of the SMN complex (Q24682443) (← links)
• Commonality amid diversity: Multi-study proteomic identification of conserved disease mechanisms in spinal muscular atrophy (Q26741505) (← links)
• A stochastic view of spliceosome assembly and recycling in the nucleus (Q27339309) (← links)
• Coilin: The first 25 years (Q28084574) (← links)
• TDP-43 expression in mouse models of amyotrophic lateral sclerosis and spinal muscular atrophy (Q33379948) (← links)
• Genetic analysis of nuclear bodies: from nondeterministic chaos to deterministic order (Q33775603) (← links)
• The role of nuclear bodies in gene expression and disease (Q34038183) (← links)
• The Cajal body and the nucleolus: "In a relationship" or "It's complicated"? (Q34047421) (← links)
• Whole-genome screening identifies proteins localized to distinct nuclear bodies (Q34377737) (← links)
• Cajal body proteins differentially affect the processing of box C/D scaRNPs (Q35391951) (← links)
• TDRD6 mediates early steps of spliceosome maturation in primary spermatocytes. (Q36298224) (← links)
• The Hsp90 chaperone controls the biogenesis of L7Ae RNPs through conserved machinery (Q36446601) (← links)
• Gemin3 is an essential gene required for larval motor function and pupation in Drosophila (Q37035061) (← links)
• The loss of the snoRNP chaperone Nopp140 from Cajal bodies of patient fibroblasts correlates with the severity of spinal muscular atrophy (Q37128359) (← links)
• Fibroblast growth factor-2 regulates the stability of nuclear bodies (Q37293368) (← links)
• Phosphorylation and the Cajal body: modification in search of function (Q37700947) (← links)
• Fam118B, a newly identified component of Cajal bodies, is required for Cajal body formation, snRNP biogenesis and cell viability (Q37733004) (← links)
• Cajal bodies: where form meets function (Q38050096) (← links)
• Gem depletion: amyotrophic lateral sclerosis and spinal muscular atrophy crossover. (Q38197233) (← links)
• Spliceosomal small nuclear ribonucleoprotein particles repeatedly cycle through Cajal bodies (Q38629976) (← links)
• UsnRNP biogenesis: mechanisms and regulation (Q38647597) (← links)
• SMN - A chaperone for nuclear RNP social occasions? (Q38959600) (← links)
• Depletion of hCINAP by RNA interference causes defects in Cajal body formation, histone transcription, and cell viability (Q39734889) (← links)
• The SMN protein is a key regulator of nuclear architecture in differentiating neuroblastoma cells. (Q39802654) (← links)
• Survival motor neuron protein regulates apoptosis in an in vitro model of spinal muscular atrophy (Q39998384) (← links)
• Post-transcriptional modification of spliceosomal RNAs is normal in SMN-deficient cells (Q40520534) (← links)
• Diabetic polyneuropathy, sensory neurons, nuclear structure and spliceosome alterations: a role for CWC22. (Q41866847) (← links)
• Changes in intranuclear mobility of mature snRNPs provide a mechanism for splicing defects in spinal muscular atrophy. (Q42144658) (← links)
• Post-transcriptional spliceosomes are retained in nuclear speckles until splicing completion (Q46686083) (← links)
• CBC-ARS2 stimulates 3'-end maturation of multiple RNA families and favors cap-proximal processing (Q50335775) (← links)
• Impaired minor tri-snRNP assembly generates differential splicing defects of U12-type introns in lymphoblasts derived from a type I SMA patient (Q52605306) (← links)
• Diabetic neuropathy and the sensory neuron: New aspects of pathogenesis and their treatment implications. (Q52658260) (← links)
• DMA-tudor interaction modules control the specificity of in vivo condensates (Q111442037) (← links)