Pages that link to "Q34549944"
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The following pages link to Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4A and p14ARF expression (Q34549944):
Displaying 50 items.
- Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis (Q26752541) (← links)
- Potential cellular functions of Epstein-Barr Nuclear Antigen 1 (EBNA1) of Epstein-Barr Virus (Q26852071) (← links)
- Epigenetic Impact on EBV Associated B-Cell Lymphomagenesis (Q28068361) (← links)
- Epstein-Barr virus latent genes (Q28082701) (← links)
- The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth (Q28546003) (← links)
- Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines (Q31111648) (← links)
- Developments in Burkitt's lymphoma: novel cooperations in oncogenic MYC signaling (Q34039568) (← links)
- Upregulation of the cell-cycle regulator RGC-32 in Epstein-Barr virus-immortalized cells (Q34097970) (← links)
- E2F1 mediated apoptosis induced by the DNA damage response is blocked by EBV nuclear antigen 3C in lymphoblastoid cells. (Q34205875) (← links)
- Epstein-Barr virus nuclear antigen 3A promotes cellular proliferation by repression of the cyclin-dependent kinase inhibitor p21WAF1/CIP1. (Q34283174) (← links)
- The effect of Epstein-Barr virus Latent Membrane Protein 2 expression on the kinetics of early B cell infection (Q34545445) (← links)
- Functional modulation of the metastatic suppressor Nm23-H1 by oncogenic viruses (Q34901718) (← links)
- Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes (Q34985686) (← links)
- Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming (Q34998243) (← links)
- Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9 (Q34998257) (← links)
- Epstein-Barr virus-positive T/NK-cell lymphoproliferative disorders (Q35040638) (← links)
- EBNA3C augments Pim-1 mediated phosphorylation and degradation of p21 to promote B-cell proliferation (Q35224422) (← links)
- Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2. (Q35685797) (← links)
- EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors (Q35858022) (← links)
- Epstein-Barr virus oncoprotein super-enhancers control B cell growth (Q35963272) (← links)
- Epigenetic silencing of tumor suppressor genes during in vitro Epstein-Barr virus infection. (Q36079181) (← links)
- Cyclin D1 overexpression supports stable EBV infection in nasopharyngeal epithelial cells (Q36483613) (← links)
- Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites (Q36736629) (← links)
- Towards incorporating epigenetic mechanisms into carcinogen identification and evaluation (Q37151661) (← links)
- Epstein-Barr virus induces global changes in cellular mRNA isoform usage that are important for the maintenance of latency (Q37254393) (← links)
- Epstein-Barr virus nuclear antigen leader protein localizes to promoters and enhancers with cell transcription factors and EBNA2. (Q37319836) (← links)
- Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A. (Q37474974) (← links)
- Epstein-Barr virus super-enhancer eRNAs are essential for MYC oncogene expression and lymphoblast proliferation (Q37493384) (← links)
- Inhibition of germinal centre apoptotic programmes by epstein-barr virus (Q37959963) (← links)
- Epigenomic deregulation in the immune system. (Q37962605) (← links)
- Impact of EBV essential nuclear protein EBNA-3C on B-cell proliferation and apoptosis (Q38086861) (← links)
- Interplay between DNA tumor viruses and the host DNA damage response. (Q38107828) (← links)
- EBV finds a polycomb-mediated, epigenetic solution to the problem of oncogenic stress responses triggered by infection (Q38156169) (← links)
- Switching of EBV cycles between latent and lytic states (Q38171455) (← links)
- Dynamic Epstein-Barr virus gene expression on the path to B-cell transformation (Q38174306) (← links)
- Modes of infection and oncogenesis by the Epstein-Barr virus. (Q38191876) (← links)
- Role of epigenetics in EBV regulation and pathogenesis (Q38232195) (← links)
- Epstein-Barr virus infection and persistence in nasopharyngeal epithelial cells (Q38249886) (← links)
- The Epstein-Barr virus and the pathogenesis of lymphoma. (Q38258049) (← links)
- At a crossroads: human DNA tumor viruses and the host DNA damage response. (Q38388893) (← links)
- The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B Cells (Q38597236) (← links)
- EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency (Q38644507) (← links)
- An essential EBV latent antigen 3C binds Bcl6 for targeted degradation and cell proliferation. (Q38664787) (← links)
- Epigenetic Alterations in Epstein-Barr Virus-Associated Diseases (Q38667123) (← links)
- Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression (Q38728190) (← links)
- Coexistence of intestinal Kaposi sarcoma and plasmablastic lymphoma in an HIV/AIDS patient: case report and review of the literature (Q38795372) (← links)
- Combination of SAHA and bortezomib up-regulates CDKN2A and CDKN1A and induces apoptosis of Epstein-Barr virus-positive Wp-restricted Burkitt lymphoma and lymphoblastoid cell lines (Q38962956) (← links)
- Latent Membrane Protein 1 (LMP1) and LMP2A Collaborate To Promote Epstein-Barr Virus-Induced B Cell Lymphomas in a Cord Blood-Humanized Mouse Model but Are Not Essential. (Q40375799) (← links)
- Mitogen-induced B-cell proliferation activates Chk2-dependent G1/S cell cycle arrest (Q41901371) (← links)
- The Epstein-Barr Virus Regulome in Lymphoblastoid Cells. (Q41917994) (← links)