Pages that link to "Q34472101"
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The following pages link to Epstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines (Q34472101):
Displaying 30 items.
- Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis (Q26752541) (← links)
- Epigenetic Impact on EBV Associated B-Cell Lymphomagenesis (Q28068361) (← links)
- Epstein-Barr virus latent genes (Q28082701) (← links)
- The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth (Q28546003) (← links)
- Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines (Q31111648) (← links)
- C-terminal region of EBNA-2 determines the superior transforming ability of type 1 Epstein-Barr virus by enhanced gene regulation of LMP-1 and CXCR7. (Q33998709) (← links)
- Epstein-Barr virus nuclear antigen 3A protein regulates CDKN2B transcription via interaction with MIZ-1 (Q34115503) (← links)
- Epstein-Barr virus nuclear antigen 3A promotes cellular proliferation by repression of the cyclin-dependent kinase inhibitor p21WAF1/CIP1. (Q34283174) (← links)
- Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming (Q34998243) (← links)
- Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9 (Q34998257) (← links)
- EBNA3C augments Pim-1 mediated phosphorylation and degradation of p21 to promote B-cell proliferation (Q35224422) (← links)
- EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors (Q35858022) (← links)
- EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells (Q35889902) (← links)
- Epstein-Barr virus oncoprotein super-enhancers control B cell growth (Q35963272) (← links)
- Epstein-Barr Virus Nuclear Antigen 3 (EBNA3) Proteins Regulate EBNA2 Binding to Distinct RBPJ Genomic Sites (Q36736629) (← links)
- MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs. (Q37217649) (← links)
- Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A. (Q37474974) (← links)
- Impact of EBV essential nuclear protein EBNA-3C on B-cell proliferation and apoptosis (Q38086861) (← links)
- EBV finds a polycomb-mediated, epigenetic solution to the problem of oncogenic stress responses triggered by infection (Q38156169) (← links)
- The EBNA3 Family: Two Oncoproteins and a Tumour Suppressor that Are Central to the Biology of EBV in B Cells (Q38597236) (← links)
- Epigenetic Alterations in Epstein-Barr Virus-Associated Diseases (Q38667123) (← links)
- Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression (Q38728190) (← links)
- An EBV recombinant deleted for residues 130-159 in EBNA3C can deregulate p53/Mdm2 and Cyclin D1/CDK6 which results in apoptosis and reduced cell proliferation (Q38791533) (← links)
- Downregulation of integrin receptor-signaling genes by Epstein-Barr virus EBNA 3C via promoter-proximal and -distal binding elements (Q39392125) (← links)
- Analysis of Epstein-Barr virus-regulated host gene expression changes through primary B-cell outgrowth reveals delayed kinetics of latent membrane protein 1-mediated NF-κB activation (Q42701290) (← links)
- An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model (Q57025486) (← links)
- EBNA3C facilitates RASSF1A downregulation through ubiquitin-mediated degradation and promoter hypermethylation to drive B-cell proliferation (Q61450799) (← links)
- Epstein-Barr virus subverts mevalonate and fatty acid pathways to promote infected B-cell proliferation and survival (Q90095448) (← links)
- RNA Sequencing Analyses of Gene Expression during Epstein-Barr Virus Infection of Primary B Lymphocytes (Q90282529) (← links)
- Modulation of Angiogenic Processes by the Human Gammaherpesviruses, Epstein-Barr Virus and Kaposi's Sarcoma-Associated Herpesvirus (Q92238951) (← links)