Pages that link to "Q34381802"
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The following pages link to Biochemical correlates of short-term sensitization in Aplysia: temporal analysis of adenylate cyclase stimulation in a perfused-membrane preparation (Q34381802):
Displaying 14 items.
- Role of Ca2+-stimulated adenylyl cyclases in LTP and memory formation (Q33948664) (← links)
- Prepuberal stimulation of 5-HT7-R by LP-211 in a rat model of hyper-activity and attention-deficit: permanent effects on attention, brain amino acids and synaptic markers in the fronto-striatal interface. (Q35141699) (← links)
- Cyclic AMP levels, adenylyl cyclase activity, and their stimulation by serotonin quantified in intact neurons (Q36435866) (← links)
- Temporal asymmetry in activation of Aplysia adenylyl cyclase by calcium and transmitter may explain temporal requirements of conditioning (Q37111715) (← links)
- Role of phosphodiesterase 5 in synaptic plasticity and memory (Q37252748) (← links)
- Signal convergence on protein kinase A as a molecular correlate of learning (Q37542011) (← links)
- Reversal of synaptic depression by serotonin at Aplysia sensory neuron synapses involves activation of adenylyl cyclase (Q37604685) (← links)
- Injury-related behavior and neuronal plasticity: an evolutionary perspective on sensitization, hyperalgesia, and analgesia. (Q40569991) (← links)
- Pharmacological characterization of an adenylyl cyclase-coupled 5-HT receptor in aplysia: comparison with mammalian 5-HT receptors (Q44336660) (← links)
- Comparative localization of two serotonin receptors and sensorin in the central nervous system of Aplysia californica (Q46636703) (← links)
- Stimulation of cAMP response element (CRE)-mediated transcription during contextual learning (Q48240067) (← links)
- Ubiquitous molecular substrates for associative learning and activity-dependent neuronal facilitation (Q48339690) (← links)
- Induction of CRE-mediated gene expression by stimuli that generate long-lasting LTP in area CA1 of the hippocampus (Q48983531) (← links)
- Persistent effects of cyclic adenosine monophosphate are directly responsible for maintaining a neural network state. (Q64993079) (← links)