Pages that link to "Q33474807"
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The following pages link to Epstein-barr virus latency in B cells leads to epigenetic repression and CpG methylation of the tumour suppressor gene Bim (Q33474807):
Displaying 50 items.
- The Modulation of Apoptotic Pathways by Gammaherpesviruses (Q26747452) (← links)
- Role of EBNA-3 Family Proteins in EBV Associated B-cell Lymphomagenesis (Q26752541) (← links)
- Regulation of Bim in Health and Disease (Q26781203) (← links)
- Early-life exposures to infectious agents and later cancer development (Q26786103) (← links)
- Homeostasis and function of regulatory T cells in aging (Q27022786) (← links)
- Epigenetic impact of infection on carcinogenesis: mechanisms and applications (Q28066519) (← links)
- Epigenetic Impact on EBV Associated B-Cell Lymphomagenesis (Q28068361) (← links)
- Epstein-Barr virus latent genes (Q28082701) (← links)
- The Epstein-Barr Virus (EBV) in T Cell and NK Cell Lymphomas: Time for a Reassessment (Q30278553) (← links)
- Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines (Q31111648) (← links)
- Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP. (Q33604848) (← links)
- Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection (Q33658144) (← links)
- Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification (Q33749934) (← links)
- Burkitt's lymphoma (Q34030708) (← links)
- Developments in Burkitt's lymphoma: novel cooperations in oncogenic MYC signaling (Q34039568) (← links)
- Epstein-Barr virus nuclear antigen 3A protein regulates CDKN2B transcription via interaction with MIZ-1 (Q34115503) (← links)
- Epstein-Barr virus nuclear antigen 3A promotes cellular proliferation by repression of the cyclin-dependent kinase inhibitor p21WAF1/CIP1. (Q34283174) (← links)
- Epigenetic silencing of Bim transcription by Spi-1/PU.1 promotes apoptosis resistance in leukaemia (Q34357441) (← links)
- Epstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines (Q34472101) (← links)
- Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4A and p14ARF expression (Q34549944) (← links)
- A major role for Bim in regulatory T cell homeostasis. (Q34731760) (← links)
- Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming (Q34998243) (← links)
- Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9 (Q34998257) (← links)
- Expression of dual-specificity phosphatase 5 pseudogene 1 (DUSP5P1) in tumor cells (Q35126260) (← links)
- Epstein-Barr virus down-regulates tumor suppressor DOK1 expression. (Q35165290) (← links)
- EBNA3C augments Pim-1 mediated phosphorylation and degradation of p21 to promote B-cell proliferation (Q35224422) (← links)
- Viral epigenetics (Q35774163) (← links)
- EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors (Q35858022) (← links)
- The Epstein-Barr Virus BART microRNAs target the pro-apoptotic protein Bim. (Q35924384) (← links)
- Hypermethylation of the tumor suppressor gene PRDM1/Blimp-1 supports a pathogenetic role in EBV-positive Burkitt lymphoma (Q36065290) (← links)
- BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV (Q36180229) (← links)
- Epstein-Barr virus interactions with the Bcl-2 protein family and apoptosis in human tumor cells (Q36526103) (← links)
- Epstein-Barr virus-induced epigenetic alterations following transient infection (Q36625981) (← links)
- Epigenetic silencing of the proapoptotic gene BIM in anaplastic large cell lymphoma through an MeCP2/SIN3a deacetylating complex. (Q36801093) (← links)
- The B cell transcription program mediates hypomethylation and overexpression of key genes in Epstein-Barr virus-associated proliferative conversion. (Q36872499) (← links)
- Epstein-Barr virus maintains lymphomas via its miRNAs. (Q36948327) (← links)
- IL-15 Fosters Age-Driven Regulatory T Cell Accrual in the Face of Declining IL-2 Levels (Q36948861) (← links)
- Towards incorporating epigenetic mechanisms into carcinogen identification and evaluation (Q37151661) (← links)
- MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs. (Q37217649) (← links)
- Higher methylation intensity induced by EBV LMP1 via NF-κB/DNMT3b signaling contributes to silencing of PTEN gene (Q37457770) (← links)
- Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A. (Q37474974) (← links)
- Inhibition of Bim enhances replication of varicella-zoster virus and delays plaque formation in virus-infected cells (Q37547526) (← links)
- Large-scale hypomethylated blocks associated with Epstein-Barr virus-induced B-cell immortalization (Q37550182) (← links)
- How does Epstein-Barr virus (EBV) complement the activation of Myc in the pathogenesis of Burkitt's lymphoma? (Q37563700) (← links)
- Epigenetic reprogramming of host genes in viral and microbial pathogenesis (Q37781197) (← links)
- Epigenetic regulation of cell life and death decisions and deregulation in cancer (Q37785993) (← links)
- Inhibition of germinal centre apoptotic programmes by epstein-barr virus (Q37959963) (← links)
- Epigenomic deregulation in the immune system. (Q37962605) (← links)
- Impact of EBV essential nuclear protein EBNA-3C on B-cell proliferation and apoptosis (Q38086861) (← links)
- BamHI-A rightward frame 1, an Epstein-Barr virus-encoded oncogene and immune modulator. (Q38133428) (← links)