Pages that link to "Q30597707"

The following pages link to A circuitry and biochemical basis for tuberous sclerosis symptoms: from epilepsy to neurocognitive deficits (Q30597707):

Displaying 26 items.

• Genetics of cognition in epilepsy (Q24289392) (← links)
• Convulsive seizures from experimental focal cortical dysplasia occur independently of cell misplacement. (Q27314858) (← links)
• FMRP S499 is phosphorylated independent of mTORC1-S6K1 activity (Q33578330) (← links)
• MEK-ERK1/2-dependent FLNA overexpression promotes abnormal dendritic patterning in tuberous sclerosis independent of mTOR. (Q34288834) (← links)
• mTOR signaling in epilepsy: insights from malformations of cortical development (Q35245211) (← links)
• Prolonging the survival of Tsc2 conditional knockout mice by glutamine supplementation (Q35278772) (← links)
• Tuberous Sclerosis: A New Frontier in Targeted Treatment of Autism (Q35811772) (← links)
• The Stress-Induced Atf3-Gelsolin Cascade Underlies Dendritic Spine Deficits in Neuronal Models of Tuberous Sclerosis Complex (Q35897734) (← links)
• Dendritic overgrowth and elevated ERK signaling during neonatal development in a mouse model of autism (Q36400474) (← links)
• Hypoxia-inducible factor-1a contributes to dendritic overgrowth in tuberous sclerosis (Q36505662) (← links)
• Epilepsy Mechanisms in Neurocutaneous Disorders: Tuberous Sclerosis Complex, Neurofibromatosis Type 1, and Sturge-Weber Syndrome (Q37707250) (← links)
• The role of mTOR signalling in neurogenesis, insights from tuberous sclerosis complex. (Q38724342) (← links)
• The Role of mGlu Receptors in Hippocampal Plasticity Deficits in Neurological and Psychiatric Disorders: Implications for Allosteric Modulators as Novel Therapeutic Strategies (Q38863882) (← links)
• Tsc1 haploinsufficiency is sufficient to increase dendritic patterning and Filamin A levels (Q39654246) (← links)
• Protein Kinase C Epsilon Activity in the Nucleus Accumbens and Central Nucleus of the Amygdala Mediates Binge Alcohol Consumption (Q41091103) (← links)
• More Than mTOR? Novel Roles for MEK-ERK1/2 and FLNA in Tuberous Sclerosis Complex (Q42063024) (← links)
• Adjunctive everolimus therapy for treatment-resistant focal-onset seizures associated with tuberous sclerosis (EXIST-3): a phase 3, randomised, double-blind, placebo-controlled study (Q46573072) (← links)
• Dendrite Growth and the Effect of Ectopic Rheb Expression on Cortical Neurons (Q49970895) (← links)
• Metabolomic studies identify changes in transmethylation and polyamine metabolism in a brain-specific mouse model of tuberous sclerosis complex. (Q52326182) (← links)
• Role of mTOR Complexes in Neurogenesis. (Q55071609) (← links)
• Exploring the neuropsychiatric spectrum using high-content functional analysis of single-cell signaling networks (Q59817475) (← links)
• FoxO Function Is Essential for Maintenance of Autophagic Flux and Neuronal Morphogenesis in Adult Neurogenesis (Q61822444) (← links)
• Neural progenitors derived from Tuberous Sclerosis Complex patients exhibit attenuated PI3K/AKT signaling and delayed neuronal differentiation (Q62868028) (← links)
• Histone deacetylase inhibitors restore normal hippocampal synaptic plasticity and seizure threshold in a mouse model of Tuberous Sclerosis Complex (Q64115751) (← links)
• Corpus Callosum White Matter Diffusivity Reflects Cumulative Neurological Comorbidity in Tuberous Sclerosis Complex (Q89231705) (← links)
• Cellular antiseizure mechanisms of everolimus in pediatric tuberous sclerosis complex, cortical dysplasia, and non-mTOR-mediated etiologies (Q90625652) (← links)