Pages that link to "Q30492548"

The following pages link to The open reading frame 3a protein of severe acute respiratory syndrome-associated coronavirus promotes membrane rearrangement and cell death (Q30492548):

Displaying 30 items.

• The role of severe acute respiratory syndrome (SARS)-coronavirus accessory proteins in virus pathogenesis (Q24605877) (← links)
• Relevance of Viroporin Ion Channel Activity on Viral Replication and Pathogenesis (Q26823195) (← links)
• Morphogenesis of Endoplasmic Reticulum Membrane-Invaginated Vesicles during Beet Black Scorch Virus Infection: Role of Auxiliary Replication Protein and New Implications of Three-Dimensional Architecture (Q27321545) (← links)
• Dependence of Golgi apparatus integrity on nitric oxide in vascular cells: implications in pulmonary arterial hypertension (Q30499465) (← links)
• The Emerging Roles of Viroporins in ER Stress Response and Autophagy Induction during Virus Infection (Q35808638) (← links)
• Molecular characterization of feline infectious peritonitis virus strain DF-2 and studies of the role of ORF3abc in viral cell tropism. (Q36023105) (← links)
• The YXXΦ motif within the severe acute respiratory syndrome coronavirus (SARS-CoV) 3a protein is crucial for its intracellular transport (Q37732056) (← links)
• Accessory proteins of SARS-CoV and other coronaviruses (Q38226293) (← links)
• Recent developments in anti-severe acute respiratory syndrome coronavirus chemotherapy (Q39727842) (← links)
• Human coronavirus NL63 open reading frame 3 encodes a virion-incorporated N-glycosylated membrane protein (Q39751469) (← links)
• Comparative sequence analysis of full-length genome of FIPV at different tissue passage levels (Q42249099) (← links)
• Role of Severe Acute Respiratory Syndrome Coronavirus Viroporins E, 3a, and 8a in Replication and Pathogenesis. (Q55056461) (← links)
• SARS-Coronavirus Open Reading Frame-3a drives multimodal necrotic cell death (Q58763483) (← links)
• Organelle dynamics and viral infections: at cross roads (Q59357968) (← links)
• The Infectious Bronchitis Coronavirus Envelope Protein Alters Golgi pH To Protect the Spike Protein and Promote the Release of Infectious Virus (Q84315396) (← links)
• Severe acute respiratory syndrome coronavirus ORF3a protein activates the NLRP3 inflammasome by promoting TRAF3-dependent ubiquitination of ASC (Q84315429) (← links)
• The ORF3a protein of SARS-CoV-2 induces apoptosis in cells (Q96576788) (← links)
• Prospects for RNAi Therapy of COVID-19 (Q98771627) (← links)
• Why do SARS-CoV-2 NSPs rush to the ER? (Q98879302) (← links)
• Syncytia formation by SARS-CoV-2 infected cells (Q100534347) (← links)
• Imperfect innate immune antagonism renders SARS-CoV-2 vulnerable towards IFN-γ and -λ (Q104529925) (← links)
• ORF3a of the COVID-19 virus SARS-CoV-2 blocks HOPS complex-mediated assembly of the SNARE complex required for autolysosome formation (Q107338320) (← links)
• Contribution of SARS-CoV-2 accessory proteins to viral pathogenicity in K18 hACE2 transgenic mice (Q107338321) (← links)
• Variations in Orf3a protein of SARS-CoV-2 alter its structure and function (Q107404382) (← links)
• Cell death mechanisms involved in cell injury caused by SARS‐CoV‐2 (Q108782685) (← links)
• ORF3a of SARS-CoV-2 promotes lysosomal exocytosis-mediated viral egress (Q109374931) (← links)
• Contribution of SARS-CoV-2 Accessory Proteins to Viral Pathogenicity in K18 Human ACE2 Transgenic Mice (Q109374960) (← links)
• Viroporins: structure, function, and their role in the life cycle of SARS-CoV-2 (Q111086228) (← links)
• A comprehensive library of fluorescent constructs of SARS‐CoV‐2 proteins and their initial characterisation in different cell types (Q111291860) (← links)
• Understanding the Role of SARS-CoV-2 ORF3a in Viral Pathogenesis and COVID-19 (Q111459296) (← links)