Pages that link to "Q28591941"
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The following pages link to Insulin-stimulated phosphorylation of the Rab GTPase-activating protein TBC1D1 regulates GLUT4 translocation (Q28591941):
Displaying 38 items.
- Protein kinase, AMP-activated, alpha 2 catalytic subunit (Q21498733) (← links)
- RUTBC1 protein, a Rab9A effector that activates GTP hydrolysis by Rab32 and Rab33B proteins (Q24318649) (← links)
- α-MSH Stimulates Glucose Uptake in Mouse Muscle and Phosphorylates Rab-GTPase-Activating Protein TBC1D1 Independently of AMPK (Q27340082) (← links)
- Whole-genome resequencing reveals loci under selection during chicken domestication (Q28275659) (← links)
- Contraction regulates site-specific phosphorylation of TBC1D1 in skeletal muscle (Q28505471) (← links)
- Deletion of Rab GAP AS160 modifies glucose uptake and GLUT4 translocation in primary skeletal muscles and adipocytes and impairs glucose homeostasis (Q30416318) (← links)
- Novel effects of Brefeldin A (BFA) in signaling through the insulin receptor (IR) pathway and regulating FoxO1-mediated transcription (Q33618341) (← links)
- TBC1D1 regulates insulin- and contraction-induced glucose transport in mouse skeletal muscle. (Q33869742) (← links)
- Regulation of the epithelial sodium channel (ENaC) by membrane trafficking (Q34065172) (← links)
- TBC proteins: GAPs for mammalian small GTPase Rab? (Q34159986) (← links)
- Ceramides and glucosylceramides are independent antagonists of insulin signaling (Q34384118) (← links)
- Signaling, cytoskeletal and membrane mechanisms regulating GLUT4 exocytosis (Q34629081) (← links)
- Roles of TBC1D1 and TBC1D4 in insulin- and exercise-stimulated glucose transport of skeletal muscle (Q34644274) (← links)
- Homozygous staggerer (sg/sg) mice display improved insulin sensitivity and enhanced glucose uptake in skeletal muscle (Q34768447) (← links)
- Mice with AS160/TBC1D4-Thr649Ala knockin mutation are glucose intolerant with reduced insulin sensitivity and altered GLUT4 trafficking. (Q34837082) (← links)
- Effects of GLUT4 expression on insulin resistance in patients with advanced liver cirrhosis (Q35148857) (← links)
- A New Member of the TBC1D15 Family from Chiloscyllium plagiosum: Rab GTPase-Activating Protein Based on Rab7 as a Substrate (Q35634173) (← links)
- Insulin and AMPK regulate FA translocase/CD36 plasma membrane recruitment in cardiomyocytes via Rab GAP AS160 and Rab8a Rab GTPase (Q35839677) (← links)
- AMPK and Exercise: Glucose Uptake and Insulin Sensitivity. (Q36628896) (← links)
- Regulatory mode shift of Tbc1d1 is required for acquisition of insulin-responsive GLUT4-trafficking activity. (Q36680563) (← links)
- Acute resistance exercise-induced IGF1 expression and subsequent GLUT4 translocation. (Q37212852) (← links)
- Rab GEFs and GAPs (Q37751479) (← links)
- Illuminating the functional and structural repertoire of human TBC/RABGAPs. (Q37976530) (← links)
- Inhibition and termination of physiological responses by GTPase activating proteins (Q37980724) (← links)
- Role of Rab GTPases and their interacting proteins in mediating metabolic signalling and regulation (Q38359174) (← links)
- Molecular control of Rab activity by GEFs, GAPs and GDI. (Q39068332) (← links)
- Live-cell single-molecule labeling and analysis of myosin motors with quantum dots (Q41023768) (← links)
- Rab28 is a TBC1D1/TBC1D4 substrate involved in GLUT4 trafficking (Q42502773) (← links)
- Increased postexercise insulin sensitivity is accompanied by increased AS160 phosphorylation in slow-twitch soleus muscle (Q43151806) (← links)
- Exploratory study on association of genetic variation in TBC1D1 with antipsychotic-induced weight gain (Q43943107) (← links)
- A Tbc1d1 Ser231Ala-knockin mutation partially impairs AICAR- but not exercise-induced muscle glucose uptake in mice. (Q50478355) (← links)
- Comprehensive functional screening of miRNAs involved in fat cell insulin sensitivity among women. (Q51107546) (← links)
- Insulin Signaling and Glucose Uptake in the Soleus Muscle of 30-Month-Old Rats After Calorie Restriction With or Without Acute Exercise. (Q55269301) (← links)
- AKT and AMPK regulate TBC1D1 through phosphorylation and its interaction with the cytosolic tail of insulin-regulated aminopeptidase IRAP (Q57056865) (← links)
- Isoform-specific AMPK association with TBC1D1 is reduced by a mutation associated with severe obesity (Q58191658) (← links)
- Exercise prevents HFD- and OVX-induced type 2 diabetes risk factors by decreasing fat storage and improving fuel utilization (Q89452067) (← links)
- Exercise and Mitochondrial Remodeling in Skeletal Muscle in Type 2 Diabetes (Q92025368) (← links)
- TBC1D1 interacting proteins, VPS13A and VPS13C, regulate GLUT4 homeostasis in C2C12 myotubes (Q100762076) (← links)