Pages that link to "Q24648909"
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The following pages link to Bok is a pro-apoptotic Bcl-2 protein with restricted expression in reproductive tissues and heterodimerizes with selective anti-apoptotic Bcl-2 family members (Q24648909):
Displaying 50 items.
- Genome-wide prediction and validation of peptides that bind human prosurvival Bcl-2 proteins (Q21145296) (← links)
- Diva, a Bcl-2 homologue that binds directly to Apaf-1 and induces BH3-independent cell death (Q22008056) (← links)
- Boo, a novel negative regulator of cell death, interacts with Apaf-1. (Q22008618) (← links)
- MCL-1S, a splicing variant of the antiapoptotic BCL-2 family member MCL-1, encodes a proapoptotic protein possessing only the BH3 domain (Q22254196) (← links)
- MAP-1, a novel proapoptotic protein containing a BH3-like motif that associates with Bax through its Bcl-2 homology domains (Q24290468) (← links)
- Regulation of cell death in human fetal and adult ovaries—Role of Bok and Bcl-XL (Q24293623) (← links)
- Adenovirus-mediated overexpression of p14(ARF) induces p53 and Bax-independent apoptosis (Q24300313) (← links)
- BOK and NOXA are essential mediators of p53-dependent apoptosis (Q24337595) (← links)
- Thyrostimulin, a heterodimer of two new human glycoprotein hormone subunits, activates the thyroid-stimulating hormone receptor (Q24552114) (← links)
- Impact of fixed-dose combination of germacrone, curdione, and furanodiene on breast cancer cell proliferation (Q24598351) (← links)
- Herpesvirus pan encodes a functional homologue of BHRF1, the Epstein-Barr virus v-Bcl-2 (Q24804111) (← links)
- Inositol 1,4,5-trisphosphate receptor-isoform diversity in cell death and survival (Q26825354) (← links)
- Anti-apoptotic BCL-2 family proteins in acute neural injury (Q26862539) (← links)
- BAX unleashed: the biochemical transformation of an inactive cytosolic monomer into a toxic mitochondrial pore (Q27002044) (← links)
- BH3 peptides induce mitochondrial fission and cell death independent of BAX/BAK (Q27349157) (← links)
- Completing the family portrait of the anti-apoptotic Bcl-2 proteins: crystal structure of human Bfl-1 in complex with Bim (Q27652276) (← links)
- The Bcl-2 protein family member Bok binds to the coupling domain of inositol 1,4,5-trisphosphate receptors and protects them from proteolytic cleavage (Q28115007) (← links)
- Placental autophagy regulation by the BOK-MCL1 rheostat (Q28117624) (← links)
- Mtd/Bok takes a swing: proapoptotic Mtd/Bok regulates trophoblast cell proliferation during human placental development and in preeclampsia (Q28119158) (← links)
- Stabilization and enhancement of the antiapoptotic activity of mcl-1 by TCTP (Q28242429) (← links)
- Inhibition of STAT3 signaling leads to apoptosis of leukemic large granular lymphocytes and decreased Mcl-1 expression (Q28346555) (← links)
- Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death (Q28363890) (← links)
- Mtd, a novel Bcl-2 family member activates apoptosis in the absence of heterodimerization with Bcl-2 and Bcl-XL (Q28504590) (← links)
- BCL-2 family member BOK promotes apoptosis in response to endoplasmic reticulum stress (Q28507379) (← links)
- MCL1 apoptosis regulator, BCL2 family member (Q28556689) (← links)
- BCL2 family apoptosis regulator BOK (Q28558034) (← links)
- Bcl2-like 1 (Q28563257) (← links)
- A splicing variant of the Bcl-2 member Bok with a truncated BH3 domain induces apoptosis but does not dimerize with antiapoptotic Bcl-2 proteins in vitro (Q28568262) (← links)
- Localization of growth differentiation factor-9 (GDF-9) mRNA and protein in rat ovaries and cDNA cloning of rat GDF-9 and its novel homolog GDF-9B (Q28576306) (← links)
- Intracellular localization of the BCL-2 family member BOK and functional implications (Q28586794) (← links)
- The Stability and Expression Level of Bok Are Governed by Binding to Inositol 1,4,5-Trisphosphate Receptors (Q28590558) (← links)
- Bax-dependent spermatogonia apoptosis is required for testicular development and spermatogenesis (Q28592903) (← links)
- Bok Is Not Pro-Apoptotic But Suppresses Poly ADP-Ribose Polymerase-Dependent Cell Death Pathways and Protects against Excitotoxic and Seizure-Induced Neuronal Injury (Q28593344) (← links)
- BOK Is a Non-canonical BCL-2 Family Effector of Apoptosis Regulated by ER-Associated Degradation (Q28593563) (← links)
- BCL2-related protein A1 (Q29524883) (← links)
- BCL2-interacting killer (Q29526614) (← links)
- Cisplatin inhibits hippocampal cell proliferation and alters the expression of apoptotic genes (Q30413236) (← links)
- The functional genome of CA1 and CA3 neurons under native conditions and in response to ischemia (Q33302690) (← links)
- Deciphering the pathways of life and death (Q33597385) (← links)
- A portrait of the Bcl-2 protein family: life, death, and the whole picture (Q33816818) (← links)
- The relevance of spontaneous- and chemically-induced alterations in testicular germ cell apoptosis to toxicology. (Q33864774) (← links)
- Tissue-specific Bcl-2 protein partners in apoptosis: An ovarian paradigm (Q33881268) (← links)
- Germ cell suicide: new insights into apoptosis during spermatogenesis (Q33910562) (← links)
- Mechanisms underlying hypoxia-induced neuronal apoptosis (Q33935219) (← links)
- Cell death control in lymphocytes. (Q34082790) (← links)
- mTORC2 is required for proliferation and survival of TSC2-null cells (Q34177457) (← links)
- Apoptosis and its clinical impact (Q34214558) (← links)
- BCL-2 family member BOK is widely expressed but its loss has only minimal impact in mice. (Q34249957) (← links)
- A BAX/BAK and cyclophilin D-independent intrinsic apoptosis pathway (Q34311770) (← links)
- Diva reduces cell death in response to oxidative stress and cytotoxicity (Q34384826) (← links)