(Q24681309)

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Lamin A-dependent nuclear defects in human aging

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title

Lamin A-dependent nuclear defects in human aging (English)

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Paola Scaffidi

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language of work or name

English

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publication date

19 May 2006

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published in

Science

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volume

312

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issue

5776

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page(s)

1059-63

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1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

7 April 2017

Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome

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PubMed Central

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

7 April 2017

Accumulation of mutant lamin A causes progressive changes in nuclear architecture in Hutchinson-Gilford progeria syndrome

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PubMed Central

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

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7 April 2017

Genomic instability in laminopathy-based premature aging

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PubMed Central

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

7 April 2017

Recurrent de novo point mutations in lamin A cause Hutchinson–Gilford progeria syndrome

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PubMed Central

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

7 April 2017

Prelamin A, Zmpste24, misshapen cell nuclei, and progeria--new evidence suggesting that protein farnesylation could be important for disease pathogenesis

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PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

28 September 2017

Accelerated ageing in mice deficient in Zmpste24 protease is linked to p53 signalling activation.

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PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

28 September 2017

Blocking protein farnesyltransferase improves nuclear blebbing in mouse fibroblasts with a targeted Hutchinson-Gilford progeria syndrome mutation.

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PubMed Central

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

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28 September 2017

Senescing human cells and ageing mice accumulate DNA lesions with unrepairable double-strand breaks

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PubMed Central

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

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28 September 2017

CHANGES IN THE NUCLEUS WITH ADVANCING AGE OF THE ORGANISM

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PubMed Central

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

28 September 2017

Lamin a truncation in Hutchinson-Gilford progeria.

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PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

28 September 2017

Lamins: building blocks or regulators of gene expression?

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PubMed Central

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

28 September 2017

An alternative splicing product of the lamin A/C gene lacks exon 10.

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PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

28 September 2017

Cellular senescence in aging primates.

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PubMed Central

reference URL

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1855250

retrieved

28 November 2018

Identifiers

DOI

10.1126/SCIENCE.1127168

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

771468

OpenCitations bibliographic resource ID

771468

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stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

771468

PubMed publication ID

16645051

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stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

771468

ResearchGate publication ID

7136310

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